2026 ICD-10-CM Diagnosis Code E87.20Acidosis, unspecified

AHRQ’s CCSR groups this code into broader clinical categories.

• Acidosis

  a pathologic condition of acid accumulation or depletion of base in the body. the two main types are respiratory acidosis and metabolic acidosis, due to metabolic acid build up.

• Acidosis, Lactic

  acidosis caused by accumulation of lactic acid more rapidly than it can be metabolized. it may occur spontaneously or in association with diseases such as diabetes mellitus; leukemia; or liver failure.

• Acidosis, Renal Tubular

  a group of genetic disorders of the kidney tubules characterized by the accumulation of metabolically produced acids with elevated plasma chloride, hyperchloremic metabolic acidosis. defective renal acidification of urine (proximal tubules) or low renal acid excretion (distal tubules) can lead to complications such as hypokalemia, hypercalcinuria with nephrolithiasis and nephrocalcinosis, and rickets.

• Acidosis, Respiratory

  respiratory retention of carbon dioxide. it may be chronic or acute.

• Diabetic Ketoacidosis

  a life-threatening complication of diabetes mellitus, primarily of type 1 diabetes mellitus with severe insulin deficiency and extreme hyperglycemia. it is characterized by ketosis; dehydration; and depressed consciousness leading to coma.

• Hypoaldosteronism

  a congenital or acquired condition of insufficient production of aldosterone by the adrenal cortex leading to diminished aldosterone-mediated synthesis of na(+)-k(+)-exchanging atpase in renal tubular cells. clinical symptoms include hyperkalemia, sodium-wasting, hypotension, and sometimes metabolic acidosis.

• Hyperkalemia

  abnormally high potassium concentration in the blood, most often due to defective renal excretion. it is characterized clinically by electrocardiographic abnormalities (elevated t waves and depressed p waves, and eventually by atrial asystole). in severe cases, weakness and flaccid paralysis may occur. (dorland, 27th ed)

• Pseudohypoaldosteronism

  a heterogeneous group of disorders characterized by renal electrolyte transport dysfunctions. congenital forms are rare autosomal disorders characterized by neonatal hypertension, hyperkalemia, increased renin activity and aldosterone concentration. the type i features hyperkalemia with sodium wasting; type ii, hyperkalemia without sodium wasting. pseudohypoaldosteronism can be the result of a defective renal electrolyte transport protein or acquired after kidney transplantation.

• Hydrops Fetalis

  abnormal accumulation of serous fluid in two or more fetal compartments, such as skin; pleura; pericardium; placenta; peritoneum; amniotic fluid. general fetal edema may be of non-immunologic origin, or of immunologic origin as in the case of erythroblastosis fetalis.

• Compensated Metabolic Acidosis|Compensated metabolic alkalosis

  the presence of metabolic acidosis despite a nearly normal ph due to a physiologic respiratory compensatory response that lowers the partial pressure of carbon dioxide (paco2) by inducing hyperventilation.

• Diabetic Ketoacidosis|DKA|DKA|Elevated ketones/Diabetic Ketoacidosis

  the metabolic condition resulted from uncontrolled diabetes mellitus, in which the shift of acid-base status of the body toward the acid side because of loss of base or retention of acids other than carbonic acid is accompanied by the accumulation of ketone bodies in body tissues and fluids.

• Hyperchloremic Acidosis|Metabolic acidosis, normal anion gap, bicarbonate losses

  metabolic acidosis with a normal serum ratio of anions to cations and replacement of bicarbonate with chloride.

• Acidosis

  an abnormally high acidity of the blood and other body tissues. acidosis can be either respiratory or metabolic.

• Acidosis

  an abnormally high acidity (excess hydrogen-ion concentration) of the blood and other body tissues.

• Acidosis, CTCAE|Acidosis|Acidosis

  a disorder characterized by abnormally high acidity (high hydrogen-ion concentration) of the blood and other body tissues.

• Autosomal Recessive Distal Renal Tubular Acidosis-4 with Hemolytic Anemia|DRTA4

  an autosomal recessive type of distal renal tubular acidosis caused by mutation(s) in the slc4a1 gene, encoding band 3 anion transport protein. additionally, it may be characterized by hemolytic anemia.

• Diabetes Mellitus due to Underlying Condition with Ketoacidosis with Coma|Diabetes mellitus due to underlying condition with ketoacidosis with coma

  evidence of diabetes mellitus due to underlying condition with ketoacidosis with coma.

• Diabetes Mellitus due to Underlying Condition with Ketoacidosis without Coma|Diabetes mellitus due to underlying condition with ketoacidosis without coma

  evidence of diabetes mellitus due to underlying condition with ketoacidosis without coma.

• Diabetic Ketoacidosis|DKA|DKA|Elevated Ketones/Diabetic Ketoacidosis

  the metabolic condition resulted from uncontrolled diabetes mellitus, in which the shift of acid-base status of the body toward the acid side because of loss of base or retention of acids other than carbonic acid is accompanied by the accumulation of ketone bodies in body tissues and fluids.

• Distal Renal Tubular Acidosis

  failure of the renal tubules of the kidney to excrete urine of sufficient acidity, resulting in metabolic acidosis.

• Drug or Chemical Induced Diabetes Mellitus with Ketoacidosis with Coma|Drug or chemical induced diabetes mellitus with ketoacidosis with coma

  evidence of drug or chemical induced diabetes mellitus with ketoacidosis with coma.

• Drug or Chemical Induced Diabetes Mellitus with Ketoacidosis without Coma|Drug or chemical induced diabetes mellitus with ketoacidosis without coma

  evidence of drug or chemical induced diabetes mellitus with ketoacidosis without coma.

• Fetal Acidosis|Fetal Acidemia|Fetal Acidemia

  an abnormally high hydrogen ion concentration (umbilical arterial blood ph less than 7.00) in fetal blood or tissue.

• Grade 1 Acidosis, CTCAE|Grade 1 Acidosis

  ph =7.3

• Grade 3 Acidosis, CTCAE|Grade 3 Acidosis

  ph <7.3

• Grade 4 Acidosis, CTCAE|Grade 4 Acidosis

  life-threatening consequences

• Grade 5 Acidosis, CTCAE|Grade 5 Acidosis

  death

• Lactic Acidosis

  metabolic acidosis characterized by the accumulation of lactate in the body. it is caused by tissue hypoxia.

• MELAS Syndrome|MELAS|Mitochondrial Encephalomyopathy, Lactic Acidosis and Stroke

  a rare progressive neurodegenerative disorder characterized by mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke-like episodes.

• Metabolic Acidosis|Acidosis|metabolic acidosis

  increased acidity in the blood secondary to acid base imbalance. causes include diabetes, kidney failure and shock.

• Osteopetrosis with Renal Tubular Acidosis|Autosomal Recessive Osteopetrosis 3|Autosomal Recessive Osteopetrosis, Type 3|Carbonic Anhydrase II Deficiency|Guibaud-Vainsel Syndrome|Marble Brain Disease|OPTB3

  a rare, autosomal recessive inherited disorder caused by mutation in the ca2 gene. it is characterized by osteopetrosis, renal tubular acidosis, and cerebral calcifications. it results in growth failure, mental retardation, and fractures.

• Other Specified Diabetes Mellitus with Ketoacidosis with Coma|Other specified diabetes mellitus with ketoacidosis with coma

  evidence of other specified diabetes mellitus with ketoacidosis with coma not specified elsewhere.

• Other Specified Diabetes Mellitus with Ketoacidosis without Coma|Other specified diabetes mellitus with ketoacidosis without coma

  evidence of other specified diabetes mellitus with ketoacidosis without coma not specified elsewhere.

• Proximal Renal Tubular Acidosis|Type II Renal Tubular Acidosis

  impairment in renal proximal tubule bicarbonate reabsorption that results in a hypokalemic hyperchloremic metabolic acidosis, which is most commonly associated with renal fanconi syndrome.

• Renal Tubular Acidosis Associated With Deafness

  renal tubular acidosis associated with sensorineural hearing loss, and which is typically associated with recessive mutations.

• Renal Tubular Acidosis|renal tubular acidosis

  the inability of the kidneys to maintain acid-base homeostasis.

• Respiratory Acidosis

  acid base imbalance resulting from an accumulation of carbon dioxide secondary to hypoventilation.

• Respiratory Acidosis|Hypercapnic Acidosis

  a condition in which the blood ph is less than normal, secondary to impaired gas exchange.

• Type 1 Diabetes Mellitus with Ketoacidosis with Coma|Type 1 diabetes mellitus with ketoacidosis with coma

  evidence of type 1 diabetes mellitus with ketoacidosis with coma.

• Type 1 Diabetes Mellitus with Ketoacidosis without Coma|Type 1 diabetes mellitus with ketoacidosis without coma

  evidence of type 1 diabetes mellitus with ketoacidosis without coma.

• Type 2 Diabetes Mellitus with Ketoacidosis with Coma|Type 2 diabetes mellitus with ketoacidosis with coma

  evidence of type 2 diabetes mellitus with ketoacidosis with coma.

• Type 2 Diabetes Mellitus with Ketoacidosis without Coma|Type 2 diabetes mellitus with ketoacidosis without coma

  evidence of type 2 diabetes mellitus with ketoacidosis without coma.

• Type I Renal Tubular Acidosis

  impairment in renal collecting tubule acid secretion that results in a hypokalemic hyperchloremic metabolic acidosis.

• Grade 1 Hyperkalemia, CTCAE|Grade 1 Hyperkalemia

  >uln-5.5 mmol/l

• Grade 2 Hyperkalemia, CTCAE|Grade 2 Hyperkalemia

  >5.5-6.0 mmol/l; intervention initiated

• Grade 3 Hyperkalemia, CTCAE|Grade 3 Hyperkalemia

  >6.0-7.0 mmol/l; hospitalization indicated

• Grade 4 Hyperkalemia, CTCAE|Grade 4 Hyperkalemia

  >7.0 mmol/l; life-threatening consequences

• Grade 5 Hyperkalemia, CTCAE|Grade 5 Hyperkalemia

  death

• Hyperkalemia

  higher than normal levels of potassium in the circulating blood; associated with kidney failure or sometimes with the use of diuretic drugs.

• Hyperkalemia, CTCAE|Hyperkalemia|Hyperkalemia

  a disorder characterized by laboratory test results that indicate an elevation in the concentration of potassium in the blood; associated with kidney failure or sometimes with the use of diuretic drugs.

• Hyperkalemic Mineralocorticoid Resistance|Chloride Shunt Syndrome|Familial Hyperkalemic Hypertension|Gordon Hyperkalemia|Mineralocorticoid Resistant Hyperkalemia|PHA Type 2|Pseudohypoaldosteronism, Type II|Spitzer-Weinstein Syndrome

  a genetically heterogynous condition characterized by hyperkalemia, hyperchloremic acidosis, low or suppressed renin activity, and normal to high concentrations of aldosterone. mutations in genes (for example wnk1 or wnk4), regulating na-cl cotransporters (ncc), na-k-cl cotransporters (nkcc2), or the renal outer medullary potassium (romk) channel have been identified as causative in this condition. the primary abnormality is thought to be a specific defect of the renal secretory mechanism for potassium, which limits the kaliuretic response to, but not the sodium and chloride reabsorptive effect of, mineralocorticoid.

• Hydrops Fetalis

  a condition characterized by fluid accumulation in two or more anatomic compartments in the fetus.

• Immune Hydrops Fetalis

  fluid accumulation in multiple fetal anatomic cavities attributable to a maternal immune response against fetal blood cell antigens.

• Non-Immune Hydrops Fetalis

  fluid accumulation in multiple fetal anatomic cavities that is of non-immune origin.

• Hyperkalemic Mineralocorticoid Resistance|Chloride Shunt Syndrome|Familial Hyperkalemic Hypertension|Gordon Hyperkalemia|Mineralocorticoid Resistant Hyperkalemia|PHA Type 2|Pseudohypoaldosteronism, Type II|Spitzer-Weinstein Syndrome

  a genetically heterogenous condition characterized by hyperkalemia, hyperchloremic acidosis, low or suppressed renin activity, and normal to high concentrations of aldosterone. mutations in genes (for example wnk1 or wnk4), regulating na-cl cotransporters (ncc), na-k-cl cotransporters (nkcc2), or the renal outer medullary potassium (romk) channel have been identified as causative in this condition. the primary abnormality is thought to be a specific defect of the renal secretory mechanism for potassium, which limits the kaliuretic response to, but not the sodium and chloride reabsorptive effect of, mineralocorticoid.

• Grade 1 Hyperkalemia, CTCAE|Grade 1 Hyperkalemia

  >uln - 5.5 mmol/l

• Grade 2 Hyperkalemia, CTCAE|Grade 2 Hyperkalemia

  >5.5 - 6.0 mmol/l; intervention initiated

• Grade 3 Hyperkalemia, CTCAE|Grade 3 Hyperkalemia

  >6.0 - 7.0 mmol/l; hospitalization indicated

Metabolic Disorders

Metabolism is the process your body uses to get or make energy from the food you eat. Food is made up of proteins, carbohydrates, and fats. Chemicals in your digestive system break the food parts down into sugars and acids, your body's fuel.

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