2026 ICD-10-CM Diagnosis Code N05.9Unspecified nephritic syndrome with unspecified morphologic changes

AHRQ’s CCSR groups this code into broader clinical categories.

• Glomerulonephritis

  inflammation of the renal glomeruli (kidney glomerulus) that can be classified by the type of glomerular injuries including antibody deposition, complement activation, cellular proliferation, and glomerulosclerosis. these structural and functional abnormalities usually lead to hematuria; proteinuria; hypertension; and renal insufficiency.

• Glomerulonephritis, IGA

  a chronic form of glomerulonephritis characterized by deposits of predominantly immunoglobulin a in the mesangial area (glomerular mesangium). deposits of complement c3 and immunoglobulin g are also often found. clinical features may progress from asymptomatic hematuria to end-stage kidney disease.

• Glomerulonephritis, Membranoproliferative

  chronic glomerulonephritis characterized histologically by proliferation of mesangial cells, increase in the mesangial extracellular matrix, and a thickening of the glomerular capillary walls. this may appear as a primary disorder or secondary to other diseases including infections and autoimmune disease systemic lupus erythematosus. various subtypes are classified by their abnormal ultrastructures and immune deposits. hypocomplementemia is a characteristic feature of all types of mpgn.

• Glomerulonephritis, Membranous

  a type of glomerulonephritis that is characterized by the accumulation of immune deposits (complement membrane attack complex) on the outer aspect of the glomerular basement membrane. it progresses from subepithelial dense deposits, to basement membrane reaction and eventual thickening of the basement membrane.

• Glomerulosclerosis, Focal Segmental

  a clinicopathological syndrome or diagnostic term for a type of glomerular injury that has multiple causes, primary or secondary. clinical features include proteinuria, reduced glomerular filtration rate, and edema. kidney biopsy initially indicates focal segmental glomerular consolidation (hyalinosis) or scarring which can progress to globally sclerotic glomeruli leading to eventual kidney failure.

• Lupus Nephritis

  glomerulonephritis associated with autoimmune disease systemic lupus erythematosus. lupus nephritis is histologically classified into 6 classes: class i - normal glomeruli, class ii - pure mesangial alterations, class iii - focal segmental glomerulonephritis, class iv - diffuse glomerulonephritis, class v - diffuse membranous glomerulonephritis, and class vi - advanced sclerosing glomerulonephritis (the world health organization classification 1982).

• Nephrosis, Lipoid

  a kidney disease with no or minimal histological glomerular changes on light microscopy and with no immune deposits. it is characterized by lipid accumulation in the epithelial cells of kidney tubules and in the urine. patients usually show nephrotic syndrome indicating the presence of proteinuria with accompanying edema.

• Avastrovirus

  a genus of astroviridae infecting avian species and often involving extra-intestinal manifestations. the type species is turkey astrovirus.

• Heymann Nephritis Antigenic Complex

  a complex of antigenic proteins obtained from the brush border of kidney tubules. it contains two principal components low density lipoprotein receptor-related protein-2 and ldl-receptor related protein-associated protein. the name of this complex is derived from researcher, dr. walter heymann, who developed an experimental model of membranous glomerulonephritis (glomerulonephritis) by injecting this antigenic complex into rats to induce an autoimmune response.

• Low Density Lipoprotein Receptor-Related Protein-2

  an ldl-receptor related protein found in the neuroepithelium and in proximal tubular cells of the kidney. it is considered a multiligand receptor in that it binds to a variety of ligands with relatively high affinity and may function in mediating the uptake and lysosomal degradation of macromolecules such as: lipoproteins; endopeptidases; and protease inhibitors.

• Nephritis

  inflammation of any part of the kidney.

• Nephritis, Hereditary

  a group of inherited conditions characterized initially by hematuria and slowly progressing to renal insufficiency. the most common form is the alport syndrome (hereditary nephritis with hearing loss) which is caused by mutations in genes for type iv collagen and defective glomerular basement membrane.

• Nephritis, Interstitial

  inflammation of the interstitial tissue of the kidney. this term is generally used for primary inflammation of kidney tubules and/or surrounding interstitium. for primary inflammation of glomerular interstitium, see glomerulonephritis. infiltration of the inflammatory cells into the interstitial compartment results in edema, increased spaces between the tubules, and tubular renal dysfunction.

• Astroviridae

  a family of rna viruses with two genera: mamastrovirus and avastrovirus. they cause gastroenteritis in humans and also infect other vertebrates.

• Kidney

  body organ that filters blood for the secretion of urine and that regulates ion concentrations.

• Diffuse Endocapillary Glomerulonephritis|Diffuse endocapillary proliferative glomerulonephritis

  glomerulonephritis characterized by endocapillary proliferation and the presence of inflammatory cells affecting 50% or more of all glomeruli.

• Glomerulonephritis, CTCAE

  a disorder characterized by inflammation of the glomerulus, usually based on renal biopsy.

• Grade 1 Glomerulonephritis, CTCAE

  asymptomatic; laboratory findings only

• Grade 1 Tubulointerstitial Nephritis, CTCAE

  laboratory findings without increase in creatinine

• Grade 2 Glomerulonephritis, CTCAE

  declining glomerular filtration rate (gfr); edema; hypertension; oral management indicated

• Grade 2 Tubulointerstitial Nephritis, CTCAE

  increase in creatinine; oral management indicated with steroid

• Grade 3 Glomerulonephritis, CTCAE

  iv therapy indicated; immunosuppressant indicated; hospitalization or prolongation of existing hospitalization indicated; limiting self-care adl or severe impact on age-appropriate normal daily activity (pediatric)

• Grade 3 Tubulointerstitial Nephritis, CTCAE

  iv therapy indicated; hospitalization or prolongation of existing hospitalization indicated; limiting self-care adl or severe impact on age-appropriate normal daily activity (pediatric)

• Grade 4 Glomerulonephritis, CTCAE

  life-threatening consequences; urgent intervention indicated; renal replacement therapy

• Grade 4 Tubulointerstitial Nephritis, CTCAE

  life-threatening consequences; urgent intervention indicated

• Grade 5 Glomerulonephritis, CTCAE

  death

• Grade 5 Tubulointerstitial Nephritis, CTCAE

  death

• Necrotizing Glomerulonephritis|NGN|Necrotizing glomerulonephritis

  necrosis of glomeruli that results from small vessel vasculitis in the kidney.

• Refractory Lupus Glomerulonephritis

  lupus glomerulonephritis that does not respond to treatment.

• Tubulointerstitial Nephritis, CTCAE

  a disorder characterized by inflammation of the renal tubule and surrounding tissues.

• Exudative Glomerulonephritis

  inflammation of the glomeruli with infiltration by polymorphonuclear leukocytes.

• Acute Diffuse Glomerulonephritis

  an acute inflammation of the glomeruli, in which all glomeruli are affected, resulting in acute renal failure.

• Acute Glomerulonephritis

  an acute inflammation of the glomeruli, generally secondary to infection or injury.

• Acute Pyelonephritis

  sudden onset pyelonephritis.

• Alport Syndrome|Alport's Syndrome|Hereditary Nephritis

  a genetic syndrome usually inherited as an x-link trait. it is caused by abnormalities in the col4a5 gene. it affects males more often than females and is characterized by hematuria, progressive renal insufficiency, hearing loss, and ocular abnormalities.

• Anti-Basement Membrane Glomerulonephritis|Anti-GBM Antibody Disease|Anti-Glomerular Basement Membrane Antibody Disease

  inflammation of the glomeruli secondary to presence of autoantibodies directed at specific antigenic targets within the glomerular basement membrane, causing hematuria, proteinuria, and impaired renal function.

• C3 Glomerulonephritis|Complement-Mediated Membranoproliferative Glomerulonephritis

  glomerulonephritis characterized by c3 accumulation with little or absent deposition of immunoglobulin, in the absence of ultrastructural electron-dense transformation seen in dense deposit disease.

• Chronic Glomerulonephritis

  a chronic, persistent inflammation of the glomeruli, which is slowly progressive, leading to impaired kidney function.

• Chronic Glomerulonephritis with Lesion of Membranous Glomerulonephritis

  a slowly progressive inflammation of the glomeruli characterized by immune complex deposits on the epithelial side of the glomerular basement membrane.

• Chronic Glomerulonephritis with Lesion of Proliferative Glomerulonephritis

  a slowly progressive inflammation of the glomeruli occurring status post infection with streptococcus.

• Chronic Pyelonephritis

  persistent pyelonephritis.

• Crescentic Glomerulonephritis

  a histopathologic term for a pattern of diseases characterized by extensive crescent formation in the glomeruli; patients present clinically with rapid deterioration of renal function, and possible progression to end-stage renal failure within weeks or months.

• Cryoglobulinemic Glomerulonephritis

  glomerulonephritis in the context of cryoglobulinemia.

• Cryoglobulinemic Glomerulonephritis Secondary to Systemic Disease

  cryoglobulinemia glomerulonephritis associated with chronic disease, often inflammatory conditions.

• Dense Deposit Disease|Membranoproliferative Glomerulonephritis Type 2|Mesangiocapillary Glomerulonephritis Type 2

  proliferative glomerulonephritis characterized by activation of the alternative complement pathway, resulting in mesangial hypercellularity, endocapillary proliferation, and glomerular basement membrane intramembranous highly electron dense deposits.

• Diffuse Endocapillary Glomerulonephritis

  glomerulonephritis characterized by endocapillary proliferation and the presence of inflammatory cells affecting 50% or more of all glomeruli.

• Diffuse Glomerulonephritis

  inflammation of the glomeruli, in which all glomeruli are affected, resulting in renal failure.

• Drug-Induced Tubulointerstitial Nephritis

  tubulointerstitial nephritis resulting from a drug exposure.

• Fibrillary Glomerulonephritis|FGN|Non-Amyloid Fibrillary Glomerulonephritis

  glomerulonephritis characterized by the presence of congo-red negative microfibrils in the mesangium and capillary walls of the glomeruli. it is associated with marked proteinuria and leads to progressive renal failure.

• Focal and Segmental Proliferative Glomerulonephritis

  glomerulonephritis characterized by proliferation of endothelial or mesangial cells, affecting the glomeruli in a focal and segmental pattern.

• Focal Embolic Glomerulonephritis

  inflammation of a specific segment of glomeruli, which is associated with subacute bacterial endocarditis, and frequently produces microscopic hematuria without azotemia.

• Focal Glomerulonephritis

  inflammation of a specific segment of glomeruli within the kidney.

• Glomerulonephritis|GLOMERULONEPHRITIS|Glomerular Nephritis

  a renal disorder characterized by damage in the glomeruli. it may be acute or chronic, focal or diffuse, and it may lead to renal failure. causes include autoimmune disorders, infections, diabetes, and malignancies.

• Goodpasture Syndrome|Goodpasture's Syndrome|Lung Purpura with Glomerulonephritis

  an autoimmune disorder characterized by pulmonary hemorrhage and glomerulonephritis. it is a hypersensitivity reaction resulting in the formation of antibodies against the pulmonary alveoli and the basement membrane of the glomeruli.

• Henoch-Schönlein Purpura Nephritis

  glomerulonephritis in the context of henoch-schönlein purpura.

• Idiopathic Crescentic Glomerulonephritis

  crescentic glomerulonephritis, the cause of which is unknown.

• IgA Nephropathy|Berger's Disease|IGA Glomerulonephritis

  a chronic autoimmune glomerulonephritis characterized by the deposition of immunoglobulin a in the mesangium of the glomeruli. it is manifested with hematuria and mild proteinuria.

• Immune Complex Glomerulonephritis

  inflammation of the glomeruli characterized by the accumulation of antibody-antigen immune complexes, resulting in glomerular damage and impaired kidney function.

• Immune Complex Mediated Membranoproliferative Glomerulonephritis|Membranoproliferative Glomerulonephritis Type I|Mesangiocapillary Glomerulonephritis Type 1

  glomerulonephritis characterized by mesangial proliferation, endocapillary proliferation, and glomerular capillary wall remodeling with immune complex deposits from classical complement pathway activation.

• Immunotactoid Glomerulopathy|GOMMID|Glomerulonephritis with Organized Monoclonal Microtubular Immunoglobulin Deposits

  glomerulonephritis characterized by the presence of congo-red negative microfibrils in the mesangium and capillary walls of the glomeruli. morphologic findings include formation of microtubules, evident on electron microscopy, that are larger than those seen in fibrillary glomerulonephritis (30-50 versus 16-24 nm in diameter). there may be some overlap in the size of fibrils found in both conditions.

• Interstitial Nephritis|INTERSTITIAL NEPHRITIS|Tubulointerstitial Nephritis

  inflammation of the renal tubules and supporting tissues of the kidney.

• Karyomegalic Interstitial Nephritis|KIN|KMIN

  an autosomal recessive condition causes by mutation(s) in the fan1 gene, encoding fanconi-associated nuclease 1. it is characterized by renal failure, interstitial fibrosis, glomerular sclerosis, and atrophic tubules.

• Low-Density Lipoprotein Receptor-Related Protein 2|Glycoprotein 330|Heymann Nephritis Antigen Homolog|LRP-2|Low Density Lipoprotein Receptor-Related Protein 2|Megalin|gp330

  low-density lipoprotein receptor-related protein 2 (4655 aa, ~200 kda) is encoded by the human lrp2 gene. this protein plays a role in receptor-mediated endocytosis.

• Lupus Glomerulonephritis|Lupus Nephritis|SLE Nephritis

  glomerulonephritis in the context of systemic lupus erythematosus.

• Macrothrombocytopenia and Granulocyte Inclusions with or without Nephritis or Sensorineural Hearing Loss

  an autosomal dominant disorder caused by mutation(s) of the myh9 gene, encoding myosin-9. clinical features include thrombocytopenia, giant platelets, and characteristic inclusions in peripheral blood leukocytes, and may be associated with other organ dysfunction. it comprises the epstein syndrome, fechtner syndrome, may-hegglin anomaly, and sebastian syndrome-- all of which were previously believed to be distinct entities.

• Membranoproliferative Glomerulonephritis Type 3 (AQ)|Mesangiocapillary Glomerulonephritis Type 3

  glomerulonephritis similar in appearance under light microscopy to membranoproliferative glomerulonephritis (mpgn) i, but with subepithelial or transmembranous and subendothelial deposits on electron microscopy.

• Membranoproliferative Glomerulonephritis|Mesangiocapillary Glomerulonephritis

  inflammation of the glomeruli characterized by deposits at the intraglomerular mesangium, resulting in thickening of the glomerular basement membrane, activation of complement, and impaired kidney function secondary to damaged glomeruli.

• Membranous Glomerulonephritis|Membranous Glomerulonephropathy|Membranous Nephropathy

  a slowly progressive inflammation of the glomeruli characterized by immune complex deposits at the glomerular basement membrane, resulting in a thickened membrane, and nephrotic syndrome.

• Membranous Lupus Nephritis

  membranous nephritis associated with systemic lupus erythematosus.

• Mesangial Proliferative Glomerulonephritis

  a focal inflammation of glomeruli secondary to mesangial cell proliferation and matrix deposition within the mesangium.

• Minimal Change Glomerulonephritis|Lipoid Nephrosis|Lipoid Nephrosis|Minimal Change Disease|Minimal Change Disease|Minimal Change Nephropathy|Nil Disease

  a glomerular disorder characterized by the electron microscopic finding of loss of podocyte foot processes. light microscopic examination does not show glomerular changes. patients present with proteinuria and nephrotic syndrome.

• Nephritis

  inflammation of renal tissue.

• Nephrotic Syndrome with Lesion of Endothelial Glomerulonephritis

  a renal functional disorder characterized by proteinuria, edema, hyperlipidemia and hypoalbuminemia. it results from damage to the renal vascular filtration apparatus. it is further characterized by an inflammatory reaction in the endovascular lining of the glomerular capillaries. sequelae may include hypertension, atherosclerosis, infection, hypercoagulablity and renal failure.

• Nephrotic Syndrome with Lesion of Hypocomplementemic Glomerulonephritis

  nephrotic syndrome caused by glomerulonephritis associated with complement deficiency. it can result from an acute post-infectious glomerulonephritis, membranoproliferative glomerulonephritis or lupus glomerulonephritis.

• Nephrotic Syndrome with Lesion of Membranoproliferative Glomerulonephritis|Nephrotic Syndrome with Lesion of Lobular Glomerulonephritis|Nephrotic Syndrome with Lesion of Mesangiocapillary Glomerulonephritis

  a renal functional disorder characterized by proteinuria, edema, hyperlipidemia and hypoalbuminemia. it results from damage to the renal vascular filtration apparatus. it is further characterized by an inflammatory reaction of the glomerular capillaries due to the proliferation of mesangial cells, expansion of the mesangial matrix and deposition of immune complexes causing subsequent thickening of the glomerular basement membrane. sequelae may include hypertension, atherosclerosis, infection, hypercoagulablity and renal failure.

• Nephrotic Syndrome with Lesion of Membranous Glomerulonephritis

  a renal functional disorder characterized by proteinuria, edema, hyperlipidemia and hypoalbuminemia. it results from damage to the renal vascular filtration apparatus. it is further characterized by an inflammatory reaction in the glomerular capillaries due to the deposition of immune complexes and subsequent thickening of the glomerular basement membrane. sequelae may include hypertension, atherosclerosis, infection, hypercoagulablity and renal failure.

• Nephrotic Syndrome with Lesion of Minimal Change Glomerulonephritis

  a renal functional disorder characterized by proteinuria, edema, hyperlipidemia and hypoalbuminemia. it results from damage to the renal vascular filtration apparatus. it is further characterized by an inflammatory reaction in the glomerular capillaries and the effacement of the surrounding epithelial cell foot processes worsening protein leakage. sequelae may include hypertension, atherosclerosis, infection, hypercoagulablity and renal failure.

• Nephrotic Syndrome with Lesion of Persistent Glomerulonephritis

  a renal functional disorder characterized by proteinuria, edema, hyperlipidemia and hypoalbuminemia. it results from damage to the renal vascular filtration apparatus. it is further characterized by a chronic inflammatory reaction in the glomerular capillaries. sequelae may include hypertension, atherosclerosis, infection, hypercoagulablity and renal failure.

• Nephrotic Syndrome with Lesion of Proliferative Glomerulonephritis

  a renal functional disorder characterized by proteinuria, edema, hyperlipidemia and hypoalbuminemia. it results from damage to the renal vascular filtration apparatus. it is further characterized by an inflammatory reaction of the glomerular capillaries due to the proliferation of mesangial cells and expansion of the mesangial matrix. sequelae may include hypertension, atherosclerosis, infection, hypercoagulablity and renal failure.

• Pauci-Immune Glomerulonephritis - ANCA Negative|Pauci-immune Glomerulonephritis - ANCA Negative

  glomerulonephritis with paucity of glomerular staining for immunoglobulins that may be accompanied by systemic, small vessel vasculitis containing no anti neutrophil cytoplasm antibody (anca).

• Pauci-Immune Glomerulonephritis - Renal Limited|Pauci-immune Glomerulonephritis - Renal Limited

  glomerulonephritis in which anti-neutrophil cytoplasm antibody (anca) is almost always present and vasculitis is limited to the kidney.

• Pauci-Immune Glomerulonephritis associated with Eosinophilic Granulomatosis with Polyangiitis|Churg-Strauss Syndrome Associated Glomerulonephritis|Pauci-immune Glomerulonephritis associated with Eosinophilic Granulomatosis with Polyangiitis

  glomerulonephritis in the context of eosinophilic-rich granulomatosis with polyangiitis, eosinophilia, asthma and commonly anti-neutrophil cytoplasmic antibody.

• Pauci-Immune Glomerulonephritis associated with Granulomatosis with Polyangiitis|Pauci-immune Glomerulonephritis associated with Granulomatosis with Polyangiitis

  glomerulonephritis in the context of granulomatosis with polyangiitis in which anti-neutrophil cytoplasm antibody (anca) is almost always present.

• Pauci-Immune Glomerulonephritis associated with Microscopic Polyangiitis|Pauci-immune Glomerulonephritis associated with Microscopic Polyangiitis

  glomerulonephritis in the context of systemic, small vessel vasculitis in which anti neutrophil cytoplasm antibody (anca) is almost always present.

• Post-Streptococcal Glomerulonephritis|Acute Proliferative Glomerulonephritis

  inflammation of the glomeruli status post infection with nephritogenic streptococci, most often group a beta hemolytic streptococcus.

• Proliferative Glomerulonephritis

  a constellation of renal disorders characterized by an increase number of cells in the glomerulus; these disorders generally present with nephrotic syndrome, and generally progress to end stage renal failure over a matter of weeks to years, depending on the etiology. examples include iga nephropathy, membranoproliferative glomerulonephritis, and rapidly progressive glomerulonephritis.

• Proliferative Glomerulonephritis with Monoclonal IgG Deposits|PGNMID

  a sub-type of monoclonal gammopathy of renal significance, characterized by restriction to a single immunoglobulin g heavy chain subclass and a single light chain isotype. light microscopy often shows an endocapillary proliferative or membranoproliferative glomerulopathy. electron microscopy reveals electron-dense subendothelial and mesangial deposits.

• Pyelonephritis|Kidney Infection

  an acute or chronic inflammatory process affecting the kidney. it is caused by bacteria and in most cases it is the result of a urinary tract infection. signs and symptoms include fever, chills, flank pain, painful and frequent urination, cloudy or bloody urine, and confusion.

• Pyelonephritis|Kidney Infection|Kidney Infection|Kidney infection|PYELONEPHRITIS

  an inflammatory process affecting the kidney. the cause is most often bacterial, but may also be fungal in nature. signs and symptoms may include fever, chills, flank pain, painful and frequent urination, cloudy or bloody urine, and confusion.

• Rapidly Progressive Glomerulonephritis|RPGN

  inflammation of the glomeruli that is characterized by a rapid loss in renal function with glomerular crescent formation observed on biopsy; it is often seen in patients with concomitant autoimmune disease, like goodpasture's syndrome or systemic lupus erythematosus.

• Subacute Glomerulonephritis

  a term that refers to glomerular damage resulting in hematuria, proteinuria, and azotemia. the histopathologic changes include rapidly progressive glomerulonephritis and membranoproliferative glomerulonephritis.

• Systemic Lupus Erythematosus Nephritis Class I|Minimal Mesangial Lupus Nephritis

  systemic lupus erythematosus nephritis that appears normal under light microscopy, but with evidence of immune deposits by immunofluorescence. (weening, jan j. et al. (2004). the classification of glomerulonephritis in systemic lupus erythematosus revisited. journal of the american society of nephrology 15(2), 241-50.)

• Systemic Lupus Erythematosus Nephritis Class II|Mesangial Proliferative Lupus Nephritis

  systemic lupus erythematosus nephritis exhibiting mesangial hypercellularity or mesangial expansion by light microscopy, with mesangial immune deposits. isolated subepithelial or subendothelial deposits may be visible by immunofluorescence or electron microscopy, but not by light microscopy. (weening, jan j. et al. (2004). the classification of glomerulonephritis in systemic lupus erythematosus revisited. journal of the american society of nephrology 15(2), 241-50.)

• Systemic Lupus Erythematosus Nephritis Class III|Focal Lupus Nephritis

  systemic lupus erythematosus nephritis with active of inactive focal, segmental or global endo- or extracapillary glomerulonephritis involving less than 50% of all glomeruli, typically with focal subendothelial immune deposits with or without mesangial alterations. (weening, jan j. et al. (2004). the classification of glomerulonephritis in systemic lupus erythematosus revisited. journal of the american society of nephrology 15(2), 241-50.)

• Systemic Lupus Erythematosus Nephritis Class IV G

  systemic lupus erythematosus nephritis, characterized by active or inactive diffuse, global endo- or extracapillary glomerulonephritis that involves 50% or more of all glomeruli. (weening, jan j. et al. (2004). the classification of glomerulonephritis in systemic lupus erythematosus revisited. journal of the american society of nephrology 15(2), 241-50.)

• Systemic Lupus Erythematosus Nephritis Class IV S

  systemic lupus erythematosus nephritis characterized by active or inactive diffuse, segmental endo- or extracapillary glomerulonephritis that involves 50% or more of all glomeruli. (weening, jan j. et al. (2004). the classification of glomerulonephritis in systemic lupus erythematosus revisited. journal of the american society of nephrology 15(2), 241-50.)

• Systemic Lupus Erythematosus Nephritis Class IV|Diffuse Lupus Nephritis

  systemic lupus erythematosus nephritis, with active or inactive diffuse, segmental or global endo- or extracapillary glomerulonephritis involving greater than or equal to 50% of all glomeruli, typically with diffuse subendothelial immune deposits, with or without mesangial alterations. (weening, jan j. et al. (2004). the classification of glomerulonephritis in systemic lupus erythematosus revisited. journal of the american society of nephrology 15(2), 241-50.)

• Systemic Lupus Erythematosus Nephritis Class V

  systemic lupus erythematosus nephritis, with global or segmental subepithelial immune deposits or their morphologic sequelae by light microscopy and by immunofluorescence or electron microscopy, with or without mesangial alterations. (weening, jan j. et al. (2004). the classification of glomerulonephritis in systemic lupus erythematosus revisited. journal of the american society of nephrology 15(2), 241-50.)

• Systemic Lupus Erythematosus Nephritis Class VI|Advanced Sclerotic Lupus Nephritis

  systemic lupus erythematosus nephritis, with 90% or more of glomeruli globally sclerosed without residual activity. (weening, jan j. et al. (2004). the classification of glomerulonephritis in systemic lupus erythematosus revisited. journal of the american society of nephrology 15(2), 241-50.)

• TINAGL1 Gene|TINAGL1|TINAGL1|Tubulointerstitial Nephritis Antigen Like 1 Gene

  this gene may play a role in the regulation of receptor-mediated signaling.

• TINAGL1 wt Allele|ARG1|Androgen-Regulated Gene 1|GIS5|LCN7|LIECG3|Lipocalin 7 Gene|OLRG2|Oxidized LDL-Responsive Gene 2|P3ECSL|PP6614|PSEC0088|TINAG-Like 1 Gene|TINAGRP|Tubulointerstitial Nephritis Antigen Like 1 wt Allele|UNQ204/PRO230

  human tinagl1 wild-type allele is located in the vicinity of 1p35.2 and is approximately 11 kb in length. this allele, which encodes tubulointerstitial nephritis antigen-like protein, may be involved in the modulation of ligand-dependent signaling.

• Tubulointerstitial Nephritis and Uveitis|TINU

  an autoimmune disorder comprising tubulointerstitial nephritis and uveitis.

• Tubulointerstitial Nephritis Antigen-Like|Glucocorticoid-Inducible Protein 5|Lipocalin 7|OLRG-2|Oxidized LDL-Responsive Gene 2 Protein|TIN Ag-Related Protein|TIN-Ag-RP|TINAG-Like Protein 1|TINAG-Related Protein|TINAGL1|Tubulointerstitial Nephritis Antigen-Related Protein

  tubulointerstitial nephritis antigen-like (467 aa, ~52 kda) is encoded by the human tinagl1 gene. this protein may play a role in the modulation of receptor signaling.

• Type I Cryoglobulinemic Glomerulonephritis

  glomerulonephritis caused by cryoglobulins which are composed of monoclonal immunoglobulins igg, iga, or igm. it occurs in patients with lymphoproliferative disorders.

• Xanthogranulomatous Pyelonephritis

  chronic, destructive infection of the kidney characterized by lipid-laden macrophages in the setting of obstruction secondary to infected renal stones, most commonly caused by proteus or escherichia coli.

• Nephritic Syndrome

  a syndrome characterized by hematuria with dysmorphic red blood cells, red blood cell casts, and proteinuria; systemic manifestations may be present, including hypertension, edema, oliguria.

Kidney Diseases

You have two kidneys, each about the size of your fist. They are near the middle of your back, just below the rib cage. Inside each kidney there are about a million tiny structures called nephrons. They filter your blood. They remove wastes and extra water, which become urine. The urine flows through tubes called ureters.

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