N05.8 - Unspecified nephritic syndrome with other morphologic changes

Approximate Synonyms

The following clinical terms are approximate synonyms or lay terms that might be used to identify the correct diagnosis code:

• Collagenofibrotic glomerulopathy
• Fibrillary glomerulonephritis
• Focal embolic nephritis syndrome
• Immune-complex glomerulonephritis
• Light chain nephropathy
• Light chain nephropathy due to plasma cell dyscrasia
• Necrosis of capillary of kidney due to glomerular disease
• Necrotizing glomerulonephritis
• Proliferative glomerulonephritis
• Recurrent proliferative glomerulonephritis
• Renal involvement in malignant disease

Clinical Information

• Immune Complex Glomerulonephritis-. inflammation of the glomeruli characterized by the accumulation of antibody-antigen immune complexes, resulting in glomerular damage and impaired kidney function.
• C3 Glomerulonephritis|Complement-Mediated Membranoproliferative Glomerulonephritis-. glomerulonephritis characterized by c3 accumulation with little or absent deposition of immunoglobulin, in the absence of ultrastructural electron-dense transformation seen in dense deposit disease.
• Chronic Glomerulonephritis with Lesion of Proliferative Glomerulonephritis-. a slowly progressive inflammation of the glomeruli occurring status post infection with streptococcus.
• Dense Deposit Disease|Membranoproliferative Glomerulonephritis Type 2|Mesangiocapillary Glomerulonephritis Type 2-. proliferative glomerulonephritis characterized by activation of the alternative complement pathway, resulting in mesangial hypercellularity, endocapillary proliferation, and glomerular basement membrane intramembranous highly electron dense deposits.
• Focal and Segmental Proliferative Glomerulonephritis-. glomerulonephritis characterized by proliferation of endothelial or mesangial cells, affecting the glomeruli in a focal and segmental pattern.
• Immune Complex Mediated Membranoproliferative Glomerulonephritis|Membranoproliferative Glomerulonephritis Type I|Mesangiocapillary Glomerulonephritis Type 1-. glomerulonephritis characterized by mesangial proliferation, endocapillary proliferation, and glomerular capillary wall remodeling with immune complex deposits from classical complement pathway activation.
• Membranoproliferative Glomerulonephritis Type 3 (AQ)|Mesangiocapillary Glomerulonephritis Type 3-. glomerulonephritis similar in appearance under light microscopy to membranoproliferative glomerulonephritis (mpgn) i, but with subepithelial or transmembranous and subendothelial deposits on electron microscopy.
• Membranoproliferative Glomerulonephritis|Mesangiocapillary Glomerulonephritis-. inflammation of the glomeruli characterized by deposits at the intraglomerular mesangium, resulting in thickening of the glomerular basement membrane, activation of complement, and impaired kidney function secondary to damaged glomeruli.
• Mesangial Proliferative Glomerulonephritis-. a focal inflammation of glomeruli secondary to mesangial cell proliferation and matrix deposition within the mesangium.
• Nephrotic Syndrome with Lesion of Membranoproliferative Glomerulonephritis|Nephrotic Syndrome with Lesion of Lobular Glomerulonephritis|Nephrotic Syndrome with Lesion of Mesangiocapillary Glomerulonephritis-. a renal functional disorder characterized by proteinuria, edema, hyperlipidemia and hypoalbuminemia. it results from damage to the renal vascular filtration apparatus. it is further characterized by an inflammatory reaction of the glomerular capillaries due to the proliferation of mesangial cells, expansion of the mesangial matrix and deposition of immune complexes causing subsequent thickening of the glomerular basement membrane. sequelae may include hypertension, atherosclerosis, infection, hypercoagulablity and renal failure.
• Nephrotic Syndrome with Lesion of Proliferative Glomerulonephritis-. a renal functional disorder characterized by proteinuria, edema, hyperlipidemia and hypoalbuminemia. it results from damage to the renal vascular filtration apparatus. it is further characterized by an inflammatory reaction of the glomerular capillaries due to the proliferation of mesangial cells and expansion of the mesangial matrix. sequelae may include hypertension, atherosclerosis, infection, hypercoagulablity and renal failure.
• Post-Streptococcal Glomerulonephritis|Acute Proliferative Glomerulonephritis-. inflammation of the glomeruli status post infection with nephritogenic streptococci, most often group a beta hemolytic streptococcus.
• Proliferative Glomerulonephritis-. a constellation of renal disorders characterized by an increase number of cells in the glomerulus; these disorders generally present with nephrotic syndrome, and generally progress to end stage renal failure over a matter of weeks to years, depending on the etiology. examples include iga nephropathy, membranoproliferative glomerulonephritis, and rapidly progressive glomerulonephritis.
• Proliferative Glomerulonephritis with Monoclonal IgG Deposits|PGNMID-. a sub-type of monoclonal gammopathy of renal significance, characterized by restriction to a single immunoglobulin g heavy chain subclass and a single light chain isotype. light microscopy often shows an endocapillary proliferative or membranoproliferative glomerulopathy. electron microscopy reveals electron-dense subendothelial and mesangial deposits.

Diagnostic Related Groups - MS-DRG Mapping

The is diagnosis code is grouped in the following groups for version MS-DRG V40.0 What are Diagnostic Related Groups?
The Diagnostic Related Groups (DRGs) are a patient classification scheme which provides a means of relating the type of patients a hospital treats. The DRGs divides all possible principal diagnoses into mutually exclusive principal diagnosis areas referred to as Major Diagnostic Categories (MDC). applicable from 10/01/2022 through 09/30/2023.

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Patient Education

Kidney Diseases

You have two kidneys, each about the size of your fist. They are near the middle of your back, just below the rib cage. Inside each kidney there are about a million tiny structures called nephrons. They filter your blood. They remove wastes and extra water, which become urine. The urine flows through tubes called ureters. It goes to your bladder, which stores the urine until you go to the bathroom.

Most kidney diseases attack the nephrons. This damage may leave kidneys unable to remove wastes. Causes can include genetic problems, injuries, or medicines. You have a higher risk of kidney disease if you have diabetes, high blood pressure, or a close family member with kidney disease. Chronic kidney disease damages the nephrons slowly over several years. Other kidney problems include:

• Cancer
• Cysts
• Stones
• Infections

Your doctor can do blood and urine tests to check if you have kidney disease. If your kidneys fail, you will need dialysis or a kidney transplant.

NIH: National Institute of Diabetes and Digestive and Kidney Diseases

Code History

• FY 2023 - No Change, effective from 10/1/2022 through 9/30/2023
• FY 2022 - No Change, effective from 10/1/2021 through 9/30/2022
• FY 2021 - No Change, effective from 10/1/2020 through 9/30/2021
• FY 2020 - No Change, effective from 10/1/2019 through 9/30/2020
• FY 2019 - No Change, effective from 10/1/2018 through 9/30/2019
• FY 2018 - No Change, effective from 10/1/2017 through 9/30/2018
• FY 2017 - No Change, effective from 10/1/2016 through 9/30/2017
• FY 2016 - New Code, effective from 10/1/2015 through 9/30/2016 (First year ICD-10-CM implemented into the HIPAA code set)