2026 ICD-10-CM Diagnosis Code E11.9Type 2 diabetes mellitus without complications

AHRQ’s CCSR groups this code into broader clinical categories.

• Acanthosis Nigricans

  a circumscribed melanosis consisting of a brown-pigmented, velvety verrucosity or fine papillomatosis appearing in the axillae and other body folds. it occurs in association with endocrine disorders, underlying malignancy, administration of certain drugs, or as in inherited disorder.

• Diabetes Complications

  conditions or pathological processes associated with the disease of diabetes mellitus. due to the impaired control of blood glucose level in diabetic patients, pathological processes develop in numerous tissues and organs including the eye, the kidney, the blood vessels, and the nerve tissue.

• Diabetes Mellitus

  a heterogeneous group of disorders characterized by hyperglycemia and glucose intolerance.

• Diabetes Mellitus, Experimental

  diabetes mellitus induced experimentally by administration of various diabetogenic agents or by pancreatectomy.

• Diabetes Mellitus, Lipoatrophic

  a type of diabetes mellitus that is characterized by severe insulin resistance and lipodystrophy. the latter may be generalized, partial, acquired, or congenital (lipodystrophy, congenital generalized).

• Diabetes Mellitus, Type 1

  a subtype of diabetes mellitus that is characterized by insulin deficiency. it is manifested by the sudden onset of severe hyperglycemia, rapid progression to diabetic ketoacidosis, and death unless treated with insulin. the disease may occur at any age, but is most common in childhood or adolescence.

• Diabetes Mellitus, Type 2

  a subclass of diabetes mellitus that is not insulin-responsive or dependent (niddm). it is characterized initially by insulin resistance and hyperinsulinemia; and eventually by glucose intolerance; hyperglycemia; and overt diabetes. type ii diabetes mellitus is no longer considered a disease exclusively found in adults. patients seldom develop ketosis but often exhibit obesity.

• Diabetes, Gestational

  diabetes mellitus induced by pregnancy but resolved at the end of pregnancy. it does not include previously diagnosed diabetics who become pregnant (pregnancy in diabetics). gestational diabetes usually develops in late pregnancy when insulin antagonistic hormones peaks leading to insulin resistance; glucose intolerance; and hyperglycemia.

• Latent Autoimmune Diabetes in Adults

  autoimmune diabetes in adults with slowly progressive pancreatic beta cell failure and the presence of circulating autoantibodies to pancreatic islets cell antigens.

• Polyendocrinopathies, Autoimmune

  autoimmune diseases affecting multiple endocrine organs. type i is characterized by childhood onset and chronic mucocutaneous candidiasis (candidiasis, chronic mucocutaneous), while type ii exhibits any combination of adrenal insufficiency (addison's disease), lymphocytic thyroiditis (thyroiditis, autoimmune;), hypoparathyroidism; and gonadal failure. in both types organ-specific antibodies against a variety of endocrine glands have been detected. the type ii syndrome differs from type i in that it is associated with hla-a1 and b8 haplotypes, onset is usually in adulthood, and candidiasis is not present.

• Insulin Resistance

  diminished effectiveness of insulin in lowering blood sugar levels: requiring the use of 200 units or more of insulin per day to prevent hyperglycemia or ketosis.

• Metabolic Syndrome

  a cluster of symptoms that are risk factors for cardiovascular diseases and type 2 diabetes mellitus. the major components of metabolic syndrome include abdominal obesity; atherogenic dyslipidemia; hypertension; hyperglycemia; insulin resistance; a proinflammatory state; and a prothrombotic (thrombosis) state.

• Pancreatectomy

  surgical removal of the pancreas. (dorland, 28th ed)

• Acanthosis Nigricans

  a melanotic cutaneous lesion that develops in the axilla and other body folds. it may be idiopathic, drug-induced, or it may be associated with the presence of an endocrine disorder or malignancy.

• Hyperandrogenism, Insulin Resistance, Acanthosis Nigricans Syndrome|HAIR-AN Syndrome

  a condition characterized by hyperandrogenism, insulin resistance, and acanthosis nigricans, typically associated with obesity in teenage girls. it is considered to be a subtype of polycystic ovarian syndrome, but may occur in male individuals. etiology is unclear, but some cases may be associated with mutations affecting the tyrosine kinase domain of the insulin receptor.

• Insulin Resistant Diabetes Mellitus with Acanthosis Nigricans and Hyperandrogenism|Type A Insulin Resistance Syndrome

  a syndrome of insulin resistance caused by mutation(s) in the insr gene, encoding the insulin receptor. this condition is characterized by a clinical triad of hyperinsulinemia, acanthosis nigricans, and hyperandrogenism without lipodystrophy. this is the least severe of a spectrum of disorders; the other two conditions are rabson-mendenhall syndrome and donohoe syndrome.

• Homeostatic Model Assessment of Insulin Resistance

  an assessment of beta-cell function and insulin resistance based on fasting blood glucose and insulin concentrations.

• Insulin Receptor Mutation - Associated Insulin Resistance Syndromes

  insulin resistance caused by inactivating mutation(s) in the insr gene encoding the insulin receptor.

• Insulin Resistance

  decreased sensitivity to circulating insulin which may result in acanthosis nigicrans, elevated insulin level or hyperglycemia.

• Insulin Resistance Measurement|INSULINR|Insulin Resistance|Insulin Resistance

  the determination of the insulin resistance (cells inability to respond to insulin) in a biological specimen.

• Insulin Resistance Syndrome

  a cluster of closely related metabolic abnormalities associated with insulin resistance that confer an increased risk of the development of type 2 diabetes and cardiovascular disease. these abnormalities may include obesity, high blood pressure, abnormal cholesterol levels, proteinuria, and/or polycystic ovary syndrome.

• Obesity-Associated Insulin Resistance

  insulin resistance associated with obesity, which may be attributed in part to impaired insulin signaling in target tissues, or impaired insulin-stimulated glucose transport due to reduced expression of the glucose transporter protein 4.

Diabetes Type 2

Type 2 diabetes is a disease in which your blood glucose, or blood sugar, levels are too high. Glucose is your main source of energy. It comes from the foods you eat. A hormone called insulin helps the glucose get into your cells to give them energy. If you have diabetes, your body doesn't make enough insulin or doesn't use insulin well.

The full article covers:

• What is type 2 diabetes?
• What causes type 2 diabetes?
• Who is at risk for type 2 diabetes?
• What are the symptoms of type 2 diabetes?
• How is type 2 diabetes diagnosed?
• What are the treatments for type 2 diabetes?
• Can type 2 diabetes be prevented?

Read the full article at MedlinePlus

Courtesy of MedlinePlus, a service of the U.S. National Library of Medicine.