2021 ICD-10-CM Code G60.0
Hereditary motor and sensory neuropathy
Valid for Submission
G60.0 is a billable diagnosis code used to specify a medical diagnosis of hereditary motor and sensory neuropathy. The code G60.0 is valid during the fiscal year 2021 from October 01, 2020 through September 30, 2021 for the submission of HIPAA-covered transactions.
The ICD-10-CM code G60.0 might also be used to specify conditions or terms like 46,xy gonadal dysgenesis, motor and sensory neuropathy syndrome, autosomal dominant charcot-marie-tooth disease type 2 due to kif5a mutation, autosomal dominant charcot-marie-tooth disease type 2 with giant axons, autosomal dominant charcot-marie-tooth disease type 2a1, autosomal dominant charcot-marie-tooth disease type 2a2 , autosomal dominant charcot-marie-tooth disease type 2b, etc.
ICD-10: | G60.0 |
Short Description: | Hereditary motor and sensory neuropathy |
Long Description: | Hereditary motor and sensory neuropathy |
Code Classification
Tabular List of Diseases and Injuries
The Tabular List of Diseases and Injuries is a list of ICD-10 codes, organized "head to toe" into chapters and sections with guidance for inclusions, exclusions, descriptions and more. The following references are applicable to the code G60.0:
Inclusion Terms
Inclusion TermsThese terms are the conditions for which that code is to be used. The terms may be synonyms of the code title, or, in the case of "other specified" codes, the terms are a list of the various conditions assigned to that code. The inclusion terms are not necessarily exhaustive. Additional terms found only in the Alphabetic Index may also be assigned to a code.
- Charcot-Marie-Tooth disease
- Déjérine-Sottas disease
- Hereditary motor and sensory neuropathy, types I-IV
- Hypertrophic neuropathy of infancy
- Peroneal muscular atrophy (axonal type) (hypertrophic type)
- Roussy-Levy syndrome
Index to Diseases and Injuries
The Index to Diseases and Injuries is an alphabetical listing of medical terms, with each term mapped to one or more ICD-10 code(s). The following references for the code G60.0 are found in the index:
- - Ataxia, ataxy, ataxic - R27.0
- - Roussy-Lévy - G60.0
- - Atrophy, atrophic (of)
- - Charcot-Marie-Tooth - G60.0
- - muscle, muscular (diffuse) (general) (idiopathic) (primary) - M62.50
- - neuropathic (peroneal) (progressive) - G60.0
- - peroneal - G60.0
- - Curvature
- - spine (acquired) (angular) (idiopathic) (incorrect) (postural) - See: Dorsopathy, deforming;
- - due to or associated with
- - Charcot-Marie-Tooth disease - See Also: subcategory M49.8; - G60.0
- - due to or associated with
- - spine (acquired) (angular) (idiopathic) (incorrect) (postural) - See: Dorsopathy, deforming;
- - Dystrophy, dystrophia
- - muscular - G71.00
- - progressive (hereditary) - G71.09
- - Charcot-Marie (-Tooth) type - G60.0
- - progressive (hereditary) - G71.09
- - muscular - G71.00
- - Neuritis (rheumatoid) - M79.2
- - Déjérine-Sottas - G60.0
- - interstitial hypertrophic progressive - G60.0
- - progressive hypertrophic interstitial - G60.0
- - Neuropathy, neuropathic - G62.9
- - Déjérine-Sottas - G60.0
- - hereditary - G60.9
- - motor and sensory (types I-IV) - G60.0
- - hypertrophic - G60.0
- - Charcot-Marie-Tooth - G60.0
- - Déjérine-Sottas - G60.0
- - interstitial progressive - G60.0
- - of infancy - G60.0
- - motor and sensory - See Also: Polyneuropathy;
- - hereditary (types I-IV) - G60.0
- - progressive
- - hypertrophic interstitial - G60.0
- - Roussy-Lévy syndrome - G60.0
Approximate Synonyms
The following clinical terms are approximate synonyms or lay terms that might be used to identify the correct diagnosis code:
- 46,XY gonadal dysgenesis, motor and sensory neuropathy syndrome
- Autosomal dominant Charcot-Marie-Tooth disease type 2 due to KIF5A mutation
- Autosomal dominant Charcot-Marie-Tooth disease type 2 with giant axons
- Autosomal dominant Charcot-Marie-Tooth disease type 2A1
- Autosomal dominant Charcot-Marie-Tooth disease type 2A2
- Autosomal dominant Charcot-Marie-Tooth disease type 2B
- Autosomal dominant Charcot-Marie-Tooth disease type 2C
- Autosomal dominant Charcot-Marie-Tooth disease type 2D
- Autosomal dominant Charcot-Marie-Tooth disease type 2E
- Autosomal dominant Charcot-Marie-Tooth disease type 2F
- Autosomal dominant Charcot-Marie-Tooth disease type 2G
- Autosomal dominant Charcot-Marie-Tooth disease type 2I
- Autosomal dominant Charcot-Marie-Tooth disease type 2J
- Autosomal dominant Charcot-Marie-Tooth disease type 2K
- Autosomal dominant Charcot-Marie-Tooth disease type 2L
- Autosomal dominant Charcot-Marie-Tooth disease type 2M
- Autosomal dominant Charcot-Marie-Tooth disease type 2N
- Autosomal dominant Charcot-Marie-Tooth disease type 2O
- Autosomal dominant Charcot-Marie-Tooth disease type 2Q
- Autosomal dominant Charcot-Marie-Tooth disease type 2U
- Autosomal dominant intermediate Charcot-Marie-Tooth disease type A
- Autosomal dominant intermediate Charcot-Marie-Tooth disease type B
- Autosomal dominant intermediate Charcot-Marie-Tooth disease type C
- Autosomal dominant intermediate Charcot-Marie-Tooth disease type D
- Autosomal dominant intermediate Charcot-Marie-Tooth disease type E
- Autosomal dominant intermediate Charcot-Marie-Tooth disease type F
- Autosomal dominant intermediate Charcot-Marie-Tooth disease with neuropathic pain
- Autosomal dominant slowed nerve conduction velocity
- Autosomal recessive Charcot-Marie-Tooth disease with hoarseness
- Autosomal recessive intermediate Charcot-Marie-Tooth disease type A
- Autosomal recessive intermediate Charcot-Marie-Tooth disease type B
- Autosomal recessive intermediate Charcot-Marie-Tooth disease type C
- Charcot-Marie-Tooth disease type 2B1
- Charcot-Marie-Tooth disease type 2B2
- Charcot-Marie-Tooth disease type 2B5
- Charcot-Marie-Tooth disease type 2H
- Charcot-Marie-Tooth disease type 2P
- Charcot-Marie-Tooth disease type 2R
- Charcot-Marie-Tooth disease type 4
- Charcot-Marie-Tooth disease type 4A
- Charcot-Marie-Tooth disease type 4B1
- Charcot-Marie-Tooth disease type 4B2
- Charcot-Marie-Tooth disease type 4B3
- Charcot-Marie-Tooth disease type 4C
- Charcot-Marie-Tooth disease type 4D
- Charcot-Marie-Tooth disease type 4E
- Charcot-Marie-Tooth disease type 4F
- Charcot-Marie-Tooth disease type 4G
- Charcot-Marie-Tooth disease type 4H
- Charcot-Marie-Tooth disease type 4J
- Charcot-Marie-Tooth disease type ID
- Charcot-Marie-Tooth disease type IE
- Charcot-Marie-Tooth disease type IF
- Charcot-Marie-Tooth disease, deafness, intellectual disability syndrome
- Charcot-Marie-Tooth disease, type IA
- Charcot-Marie-Tooth disease, type IB
- Charcot-Marie-Tooth disease, type IC
- Charcot-Marie-Tooth disease, type II
- Charcot-Marie-Tooth Neuropathy Type 4
- Déjérine-Sottas disease
- Distal hereditary motor neuropathy type 7
- Hereditary hypertrophic neuropathy with paraproteinemia
- Hereditary liability to pressure palsies
- Hereditary motor and sensory neuropathy
- Hereditary motor and sensory neuropathy Okinawa type
- Hereditary motor and sensory neuropathy with acrodystrophy
- Hereditary motor and sensory neuropathy with optic atrophy
- Hereditary motor and sensory neuropathy with retinitis pigmentosa
- Hereditary sensorimotor neuropathy with hyperelastic skin
- Hereditary sensory and autonomic neuropathy type 1B
- Hereditary sensory and autonomic neuropathy type I
- Hereditary sensory and autonomic neuropathy type II
- Hereditary sensory and autonomic neuropathy with deafness and global delay
- Hereditary sensory-motor neuropathy, type I
- Hereditary thermosensitive neuropathy
- Hypertrophic interstitial neuropathy
- Microcephalus, complex motor and sensory axonal neuropathy syndrome
- Mixed sensory-motor polyneuropathy
- Motor polyneuropathy
- Neuropathic pain
- Roussy-Lévy syndrome
- Sensory polyneuropathy
- Severe early-onset axonal neuropathy due to mitofusin 2 deficiency
- SURF1-related Charcot-Marie-Tooth disease type 4
- X-linked Charcot-Marie-Tooth disease type 1
- X-linked Charcot-Marie-Tooth disease type 2
- X-linked Charcot-Marie-Tooth disease type 3
- X-linked Charcot-Marie-Tooth disease type 4
- X-linked Charcot-Marie-Tooth disease type 5
- X-linked Charcot-Marie-Tooth disease type 6
- X-linked hereditary motor and sensory neuropathy
- Young adult-onset distal hereditary motor neuropathy
Clinical Information
- CHARCOT MARIE TOOTH DISEASE-. a hereditary motor and sensory neuropathy transmitted most often as an autosomal dominant trait and characterized by progressive distal wasting and loss of reflexes in the muscles of the legs and occasionally involving the arms. onset is usually in the second to fourth decade of life. this condition has been divided into two subtypes hereditary motor and sensory neuropathy hmsn types i and ii. hmsn i is associated with abnormal nerve conduction velocities and nerve hypertrophy features not seen in hmsn ii. adams et al. principles of neurology 6th ed p1343
- REFSUM DISEASE-. an autosomal recessive familial disorder that usually presents in childhood with polyneuropathy; sensorineural hearing loss; ichthyosis; ataxia; retinitis pigmentosa; and cardiomyopathies. from joynt clinical neurology 1991 ch37 p58 9; rev med interne 1996;175:391 8 this condition can be caused by mutation in the genes encoding peroxisomal phytanoyl coa hydroxylase or proteins associated peroxisomal membrane leading to impaired catabolism of phytanic acid in peroxisomes.
- HEREDITARY SENSORY AND MOTOR NEUROPATHY-. a group of slowly progressive inherited disorders affecting motor and sensory peripheral nerves. subtypes include hmsns i vii. hmsn i and ii both refer to charcot marie tooth disease. hmsn iii refers to hypertrophic neuropathy of infancy. hmsn iv refers to refsum disease. hmsn v refers to a condition marked by a hereditary motor and sensory neuropathy associated with spastic paraplegia see spastic paraplegia hereditary. hmsn vi refers to hmsn associated with an inherited optic atrophy optic atrophies hereditary and hmsn vii refers to hmsn associated with retinitis pigmentosa. from adams et al. principles of neurology 6th ed p1343
- SPASTIC PARAPLEGIA HEREDITARY-. a group of inherited diseases that share similar phenotypes but are genetically diverse. different genetic loci for autosomal recessive autosomal dominant and x linked forms of hereditary spastic paraplegia have been identified. clinically patients present with slowly progressive distal limb weakness and lower extremity spasticity. peripheral sensory neurons may be affected in the later stages of the disease. j neurol neurosurg psychiatry 1998 jan;641:61 6; curr opin neurol 1997 aug;104:313 8
Convert G60.0 to ICD-9 Code
The General Equivalency Mapping (GEM) crosswalk indicates an approximate mapping between the ICD-10 code G60.0 its ICD-9 equivalent. The approximate mapping means there is not an exact match between the ICD-10 code and the ICD-9 code and the mapped code is not a precise representation of the original code.
Information for Patients
Charcot-Marie-Tooth Disease
Also called: Hereditary motor and sensory neuropathy, Peroneal muscular atrophy
Charcot-Marie-Tooth disease (CMT) is a group of genetic nerve disorders. It is named after the three doctors who first identified it. In the United States, CMT affects about 1 in 2,500 people.
CMT affects your peripheral nerves. Peripheral nerves carry movement and sensation signals between the brain and spinal cord and the rest of the body. Symptoms usually start around the teen years. Foot problems such as high arches or hammertoes can be early symptoms. As CMT progresses, your lower legs may weaken. Later, your hands may also become weak.
Doctors diagnose CMT by doing a neurologic exam, nerve tests, genetic tests, or a nerve biopsy. There is no cure. The disease can be so mild you don't realize you have it or severe enough to make you weak. Physical therapy, occupational therapy, braces and other devices and sometimes surgery can help.
NIH: National Institute of Neurological Disorders and Stroke
- Charcot-Marie-Tooth disease (Medical Encyclopedia)
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Charcot-Marie-Tooth disease Charcot-Marie-Tooth disease encompasses a group of disorders called hereditary sensory and motor neuropathies that damage the peripheral nerves. Peripheral nerves connect the brain and spinal cord to muscles and to sensory cells that detect sensations such as touch, pain, heat, and sound. Damage to the peripheral nerves that worsens over time can result in alteration or loss of sensation and wasting (atrophy) of muscles in the feet, legs, and hands.Charcot-Marie-Tooth disease usually becomes apparent in adolescence or early adulthood, but onset may occur anytime from early childhood through late adulthood. Symptoms of Charcot-Marie-Tooth disease vary in severity and age of onset even among members of the same family. Some people never realize they have the disorder because their symptoms are so mild, but most have a moderate amount of physical disability. A small percentage of people experience severe weakness or other problems which, in very rare cases, can be life-threatening. In most affected individuals, however, Charcot-Marie-Tooth disease does not affect life expectancy.Typically, the earliest symptoms of Charcot-Marie-Tooth disease result from muscle atrophy in the feet. Affected individuals may have foot abnormalities such as high arches (pes cavus), flat feet (pes planus), or curled toes (hammer toes). They often have difficulty flexing the foot or walking on the heel of the foot. These difficulties may cause a higher than normal step (steppage gait) and increase the risk of ankle injuries and tripping. As the disease worsens, muscles in the lower legs usually weaken, but leg and foot problems rarely require the use of a wheelchair.Affected individuals may also develop weakness in the hands, causing difficulty with daily activities such as writing, fastening buttons, and turning doorknobs. People with Charcot-Marie-Tooth disease typically experience a decreased sensitivity to touch, heat, and cold in the feet and lower legs, but occasionally feel aching or burning sensations. In rare cases, affected individuals have loss of vision or gradual hearing loss that sometimes leads to deafness.There are several types of Charcot-Marie-Tooth disease, which are differentiated by their effects on nerve cells and patterns of inheritance. Type 1 (CMT1) is characterized by abnormalities in myelin, the fatty substance that covers nerve cells, protecting them and helping to transmit nerve impulses. These abnormalities slow the transmission of nerve impulses and can affect the health of the nerve fiber. Type 2 (CMT2) is characterized by abnormalities in the fiber, or axon, that extends from a nerve cell body to muscles or to sense organs. These abnormalities reduce the strength of the nerve impulse. In forms of Charcot-Marie-Tooth disease classified as intermediate type, the nerve impulses are both slowed and reduced in strength, probably due to abnormalities in both myelin and axons. Type 4 (CMT4) is distinguished from the other types by its pattern of inheritance; it can affect either the axons or the myelin. Type X Charcot-Marie-Tooth disease (CMTX) is caused by mutations in genes on the X chromosome, one of the two sex chromosomes. Within the various types of Charcot-Marie-Tooth disease, subtypes (such as CMT1A, CMT1B, CMT2A, CMT4A, and CMTX1) indicate different genetic causes.Sometimes other, historical names are used to refer to particular forms of Charcot-Marie-Tooth disease. For example, Roussy-Levy syndrome is a form of CMT11 with the additional feature of rhythmic shaking (tremors). Dejerine-Sottas syndrome is a term sometimes used to describe a severe, early childhood form of Charcot-Marie-Tooth disease; it is also sometimes called type 3 (CMT3). Depending on the specific gene that is altered, this severe, early-onset form of the disorder may also be classified as CMT1 or CMT4. CMTX5 is also known as Rosenberg-Chutorian syndrome.
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Code History
- FY 2021 - No Change, effective from 10/1/2020 through 9/30/2021
- FY 2020 - No Change, effective from 10/1/2019 through 9/30/2020
- FY 2019 - No Change, effective from 10/1/2018 through 9/30/2019
- FY 2018 - No Change, effective from 10/1/2017 through 9/30/2018
- FY 2017 - No Change, effective from 10/1/2016 through 9/30/2017
- FY 2016 - New Code, effective from 10/1/2015 through 9/30/2016 (First year ICD-10-CM implemented into the HIPAA code set)