2026 ICD-10-CM Diagnosis Code P84Other problems with newborn
ICD-10-CM Codes›P00–P96›P84›P84
- Billable — Valid for Submission
- Not Chronic
P84 is a billable ICD-10-CM diagnosis code for other problems with newborn. It is valid on HIPAA claims for fiscal year 2026 (October 1, 2025 through September 30, 2026). In AHRQ's Clinical Classifications Software (CCSR), this diagnosis falls under Neonatal acidemia and hypoxia.
Code Identity
Code Classification
Approximate SynonymsGuidance
Alternate terms and clinical phrases that map to this code.
- Acidemia
- Acidosis
- Acute anoxic encephalopathy
- Anoxia of brain
- Anoxia, in liveborn infant
- Anoxic brain damage complication
- Anoxic encephalopathy
- Anoxic encephalopathy due to asphyxiation
- Anoxic encephalopathy due to cardiac arrest
- Anoxic encephalopathy due to respiratory arrest
- Anoxic-ischemic encephalopathy
- Arterial oxygen concentration below reference range
- Asphyxia in liveborn infant
- Asphyxiation
- Birth asphyxia
- Birth asphyxia with Apgar score 5 minute Apgar score 4-6
- Birth asphyxia with metabolic acidemia of cord blood
- Birth examination abnormal - for observation
- Birth examination abnormal - on treatment
- Birth examination abnormal - referred
- Blood gases outside reference range
- Blood oxygen concentration below reference range
- Blood oxygen level outside reference range
- Brain disorder resulting from a period of impaired oxygen delivery to the brain
- Cerebral anoxia complication
- Chest movement appearance finding
- Chest movement unequal
- Chorea due to cerebral anoxia
- Chorea due to inherited organic acidemia
- Chorea due to metabolic disorder
- Chronic partial asphyxia of newborn
- Chronic respiratory disease originating in perinatal period
- Compensated acidosis
- Compensated metabolic acidosis
- Compensated respiratory acidosis
- Delayed post-hypoxic leukoencephalopathy
- Desaturation of blood
- D-lactic acidosis
- Dystonia due to cerebral anoxia
- Failure of transfer of passive immunity in newborn
- Fatal infantile lactic acidosis with methylmalonic aciduria
- Fetal acidosis
- Fetal anemia
- Fetal distress, in liveborn infant
- Fetal intrauterine distress first noted during labor AND/OR delivery in liveborn infant
- Fetal intrauterine distress noted before labor in liveborn infant
- Fetal metabolic disorder
- Finding of arterial oxygen concentration
- Hydrops fetalis
- Hydrops, lactic acidosis, sideroblastic anemia, multisystemic failure syndrome
- Hypertrophic cardiomyopathy with hypotonia and lactic acidosis syndrome
- Hypertrophic mitochondrial cardiomyopathy
- Hypoxemia
- Hypoxemia during surgery
- Hypoxia
- Hypoxia of brain
- Hypoxia with feeding in newborn
- Hypoxia, in liveborn infant
- Hypoxic-ischemic coma
- Impaired gas exchange
- Impaired spontaneous ventilation
- Ineffective airway clearance
- Ineffective breathing pattern
- Infant behavior alteration
- Inhalation of liquid in lower respiratory tract
- Jittery newborn
- Left side of chest moves less than right
- Liveborn with labor fetal distress
- Liveborn with labor hypoxia
- Liveborn with prelabor fetal distress
- Liveborn with prelabor hypoxia
- Metabolic acidemia in newborn
- Metabolic acidosis
- Metabolic acidosis and metabolic alkalosis
- Metabolic acidosis, IAG, accumulation of organic acids
- Metabolic acidosis, IAG, reduced excretion of inorganic acids
- Metabolic acidosis, increased anion gap
- Metabolic acidosis, NAG, acidifying salts
- Metabolic acidosis, NAG, bicarbonate losses
- Metabolic acidosis, NAG, failure of bicarbonate regeneration
- Metabolic acidosis, normal anion gap
- Mild birth asphyxia
- Mild birth asphyxia, Apgar 4-7
- Mild to moderate birth asphyxia
- Mild to moderate birth asphyxia - Apgar score 4-7 at 1 minute
- Mitochondrial DNA depletion syndrome encephalomyopathic form
- Mixed acid-base balance disorder
- Mixed acid-base balance disorders - not compensated primary disorder
- Moderate birth asphyxia
- Neonatal acidosis
- Neonatal compression of brain
- Neonatal disorder
- Neonatal hypoxemia
- Neonatal metabolic acidemia
- Neonatal respiratory acidosis
- Neonatal respiratory alkalosis
- Newborn behavior alteration
- Obstructive ventilatory defect
- Oxygen supply absent
- Perinatal anoxic-ischemic brain injury
- Perinatal asphyxia
- Perinatal hypoxia
- Post-anoxic myoclonus
- Post-asphyxial encephalopathy
- Respiratory acidosis
- Respiratory alkalosis
- Respiratory alkalosis and metabolic acidosis
- Respiratory alteration
- Right side of chest moves less than left
- Severe birth asphyxia
- Severe birth asphyxia - Apgar score less than 4 at 1 minute
- Severe birth asphyxia, Apgar 0-3
- Severe neonatal lactic acidosis due to NFS1-ISD11 complex deficiency
- Suffocating
- Symmetry of chest movement - finding
- Unable to breathe
- Ventilatory defect
Tabular List NotesGuidance
Coding notes and annotation back-references applicable to this code.
Inclusion Terms
- Acidemia of newborn
- Acidosis of newborn
- Anoxia of newborn NOS
- Asphyxia of newborn NOS
- Hypercapnia of newborn
- Hypoxemia of newborn
- Hypoxia of newborn NOS
- Mixed metabolic and respiratory acidosis of newborn
Type 1 Excludes
- intracranial hemorrhage due to anoxia or hypoxia P52
- hypoxic ischemic encephalopathy HIE P91.6
- late metabolic acidosis of newborn P74.0
These terms are the conditions for which that code is to be used. The terms may be synonyms of the code title, or, in the case of "other specified" codes, the terms are a list of the various conditions assigned to that code. The inclusion terms are not necessarily exhaustive. Additional terms found only in the Alphabetic Index may also be assigned to a code.
A type 1 excludes note is a pure excludes note. It means "NOT CODED HERE!" An Excludes1 note indicates that the code excluded should never be used at the same time as the code above the Excludes1 note. An Excludes1 is used when two conditions cannot occur together, such as a congenital form versus an acquired form of the same condition.
Index to Diseases and InjuriesGuidance
Alphabetical index entries that point to this code.
- newborn - P84
- Anoxia (pathological) - R09.02
- newborn - P84
- intrauterine - P84
- newborn - P84
- Hypercapnia - R06.89
- newborn - P84
- Hypoxia - See Also: Anoxia; - R09.02
- intrauterine - P84
- newborn - P84
External Cause of Injuries IndexGuidance
References for this code in the External Cause of Injuries Index.
- Acidosis(lactic)
- mixed metabolic and respiratory, newborn
- Acidosis(lactic)
- newborn
- Anoxemia
- newborn
- Anoxia(pathological)
- cerebral
- newborn
- Anoxia(pathological)
- intrauterine
- Anoxia(pathological)
- newborn
- Asphyxia, asphyxiation(by)
- antenatal
- Asphyxia, asphyxiation(by)
- birth
- Asphyxia, asphyxiation(by)
- intrauterine
- Asphyxia, asphyxiation(by)
- newborn
- Asphyxia, asphyxiation(by)
- postnatal
- Asphyxia, asphyxiation(by)
- prenatal
- Distress
- fetal
- Hypercapnia
- newborn
- Hypoxemia
- newborn
- Hypoxia
- intrauterine
- Hypoxia
- newborn
- Newborn(infant) (liveborn) (singleton)
- affected by
- mixed metabolic and respiratory acidosis
- Stress
- fetal
Clinical ClassificationClinical
AHRQ’s CCSR groups this code into broader clinical categories.
Clinical InformationClinical
Hydrops Fetalis
abnormal accumulation of serous fluid in two or more fetal compartments, such as skin; pleura; pericardium; placenta; peritoneum; amniotic fluid. general fetal edema may be of non-immunologic origin, or of immunologic origin as in the case of erythroblastosis fetalis.Altitude Sickness
multiple symptoms associated with reduced oxygen at high altitude.Aryl Hydrocarbon Receptor Nuclear Translocator
aryl hydrocarbon receptor nuclear translocator is a basic helix-loop-helix motif containing protein that forms a complex with dioxin receptor. the complex binds xenobiotic regulatory elements and activates transcription of a variety of genes including udp glucuronosyltransferase. ahr nuclear translocator is also a subunit of hypoxia-inducible factor 1.Cell Hypoxia
a condition of decreased oxygen content at the cellular level.Fetal Hypoxia
deficient oxygenation of fetal blood.Hypoxia
sub-optimal oxygen levels in the ambient air of living organisms.Hypoxia, Brain
a reduction in brain oxygen supply due to anoxemia (a reduced amount of oxygen being carried in the blood by hemoglobin), or to a restriction of the blood supply to the brain, or both. severe hypoxia is referred to as anoxia and is a relatively common cause of injury to the central nervous system. prolonged brain anoxia may lead to brain death or a persistent vegetative state. histologically, this condition is characterized by neuronal loss which is most prominent in the hippocampus; globus pallidus; cerebellum; and inferior olives.Hypoxia-Inducible Factor 1
a basic helix-loop-helix transcription factor that plays a role in apoptosis. it is composed of two subunits: aryl hydrocarbon receptor nuclear translocator and hypoxia-inducible factor 1, alpha subunit.Hypoxia-Inducible Factor 1, alpha Subunit
hypoxia-inducible factor 1, alpha subunit is a basic helix-loop-helix transcription factor that is regulated by oxygen availability and is targeted for degradation by vhl tumor suppressor protein.Hypoxia-Inducible Factor-Proline Dioxygenases
dioxygenase enzymes that specifically hydroxylate a proline residue on the hypoxia-inducible factor 1, alpha subunit. they are oxygen-dependent enzymes that play an important role in mediating cellular adaptive responses to hypoxia.Hypoxia-Ischemia, Brain
a disorder characterized by a reduction of oxygen in the blood combined with reduced blood flow (ischemia) to the brain from a localized obstruction of a cerebral artery or from systemic hypoperfusion. prolonged hypoxia-ischemia is associated with ischemic attack, transient; brain infarction; brain edema; coma; and other conditions.Tumor Hypoxia
hypoxic conditions in tumor cells due to the tumor outgrowing its blood supply. it is associated with increased metastasis and resistance to radiotherapy and drug therapy.Acidosis
a pathologic condition of acid accumulation or depletion of base in the body. the two main types are respiratory acidosis and metabolic acidosis, due to metabolic acid build up.Acidosis, Lactic
acidosis caused by accumulation of lactic acid more rapidly than it can be metabolized. it may occur spontaneously or in association with diseases such as diabetes mellitus; leukemia; or liver failure.Acidosis, Renal Tubular
a group of genetic disorders of the kidney tubules characterized by the accumulation of metabolically produced acids with elevated plasma chloride, hyperchloremic metabolic acidosis. defective renal acidification of urine (proximal tubules) or low renal acid excretion (distal tubules) can lead to complications such as hypokalemia, hypercalcinuria with nephrolithiasis and nephrocalcinosis, and rickets.Acidosis, Respiratory
respiratory retention of carbon dioxide. it may be chronic or acute.Diabetic Ketoacidosis
a life-threatening complication of diabetes mellitus, primarily of type 1 diabetes mellitus with severe insulin deficiency and extreme hyperglycemia. it is characterized by ketosis; dehydration; and depressed consciousness leading to coma.Hypoaldosteronism
a congenital or acquired condition of insufficient production of aldosterone by the adrenal cortex leading to diminished aldosterone-mediated synthesis of na(+)-k(+)-exchanging atpase in renal tubular cells. clinical symptoms include hyperkalemia, sodium-wasting, hypotension, and sometimes metabolic acidosis.Altitude
a vertical distance measured from a known level on the surface of a planet or other celestial body.Fetal Blood
blood of the fetus. exchange of nutrients and waste between the fetal and maternal blood occurs via the placenta. the cord blood is blood contained in the umbilical vessels (umbilical cord) at the time of delivery.Oxygen
an element with atomic symbol o, atomic number 8, and atomic weight [15.99903; 15.99977]. it is the most abundant element on earth and essential for respiration.Compensated Metabolic Acidosis|Compensated metabolic alkalosis
the presence of metabolic acidosis despite a nearly normal ph due to a physiologic respiratory compensatory response that lowers the partial pressure of carbon dioxide (paco2) by inducing hyperventilation.Diabetic Ketoacidosis|DKA|DKA|Elevated ketones/Diabetic Ketoacidosis
the metabolic condition resulted from uncontrolled diabetes mellitus, in which the shift of acid-base status of the body toward the acid side because of loss of base or retention of acids other than carbonic acid is accompanied by the accumulation of ketone bodies in body tissues and fluids.Hyperchloremic Acidosis|Metabolic acidosis, normal anion gap, bicarbonate losses
metabolic acidosis with a normal serum ratio of anions to cations and replacement of bicarbonate with chloride.Respiratory Alkalosis
a condition in which the blood ph is greater than normal, secondary to impaired gas exchange.Respiratory Acidosis
acid base imbalance resulting from an accumulation of carbon dioxide secondary to hypoventilation.Respiratory Acidosis|Hypercapnic Acidosis
a condition in which the blood ph is less than normal, secondary to impaired gas exchange.Neonatal Disorder
a non-neoplastic or neoplastic disorder which occurs during the neonatal period.Hydrops Fetalis
a condition characterized by fluid accumulation in two or more anatomic compartments in the fetus.Immune Hydrops Fetalis
fluid accumulation in multiple fetal anatomic cavities attributable to a maternal immune response against fetal blood cell antigens.Non-Immune Hydrops Fetalis
fluid accumulation in multiple fetal anatomic cavities that is of non-immune origin.Acidosis
an abnormally high acidity of the blood and other body tissues. acidosis can be either respiratory or metabolic.Acidosis
an abnormally high acidity (excess hydrogen-ion concentration) of the blood and other body tissues.Acidosis, CTCAE|Acidosis|Acidosis
a disorder characterized by abnormally high acidity (high hydrogen-ion concentration) of the blood and other body tissues.Autosomal Recessive Distal Renal Tubular Acidosis-4 with Hemolytic Anemia|DRTA4
an autosomal recessive type of distal renal tubular acidosis caused by mutation(s) in the slc4a1 gene, encoding band 3 anion transport protein. additionally, it may be characterized by hemolytic anemia.Diabetes Mellitus due to Underlying Condition with Ketoacidosis with Coma|Diabetes mellitus due to underlying condition with ketoacidosis with coma
evidence of diabetes mellitus due to underlying condition with ketoacidosis with coma.Diabetes Mellitus due to Underlying Condition with Ketoacidosis without Coma|Diabetes mellitus due to underlying condition with ketoacidosis without coma
evidence of diabetes mellitus due to underlying condition with ketoacidosis without coma.Diabetic Ketoacidosis|DKA|DKA|Elevated Ketones/Diabetic Ketoacidosis
the metabolic condition resulted from uncontrolled diabetes mellitus, in which the shift of acid-base status of the body toward the acid side because of loss of base or retention of acids other than carbonic acid is accompanied by the accumulation of ketone bodies in body tissues and fluids.Distal Renal Tubular Acidosis
failure of the renal tubules of the kidney to excrete urine of sufficient acidity, resulting in metabolic acidosis.Drug or Chemical Induced Diabetes Mellitus with Ketoacidosis with Coma|Drug or chemical induced diabetes mellitus with ketoacidosis with coma
evidence of drug or chemical induced diabetes mellitus with ketoacidosis with coma.Drug or Chemical Induced Diabetes Mellitus with Ketoacidosis without Coma|Drug or chemical induced diabetes mellitus with ketoacidosis without coma
evidence of drug or chemical induced diabetes mellitus with ketoacidosis without coma.Fetal Acidosis|Fetal Acidemia|Fetal Acidemia
an abnormally high hydrogen ion concentration (umbilical arterial blood ph less than 7.00) in fetal blood or tissue.Grade 1 Acidosis, CTCAE|Grade 1 Acidosis
ph=7.3 Grade 3 Acidosis, CTCAE|Grade 3 Acidosis
ph <7.3Grade 4 Acidosis, CTCAE|Grade 4 Acidosis
life-threatening consequencesGrade 5 Acidosis, CTCAE|Grade 5 Acidosis
deathLactic Acidosis
metabolic acidosis characterized by the accumulation of lactate in the body. it is caused by tissue hypoxia.MELAS Syndrome|MELAS|Mitochondrial Encephalomyopathy, Lactic Acidosis and Stroke
a rare progressive neurodegenerative disorder characterized by mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke-like episodes.Metabolic Acidosis|Acidosis|metabolic acidosis
increased acidity in the blood secondary to acid base imbalance. causes include diabetes, kidney failure and shock.Osteopetrosis with Renal Tubular Acidosis|Autosomal Recessive Osteopetrosis 3|Autosomal Recessive Osteopetrosis, Type 3|Carbonic Anhydrase II Deficiency|Guibaud-Vainsel Syndrome|Marble Brain Disease|OPTB3
a rare, autosomal recessive inherited disorder caused by mutation in the ca2 gene. it is characterized by osteopetrosis, renal tubular acidosis, and cerebral calcifications. it results in growth failure, mental retardation, and fractures.Other Specified Diabetes Mellitus with Ketoacidosis with Coma|Other specified diabetes mellitus with ketoacidosis with coma
evidence of other specified diabetes mellitus with ketoacidosis with coma not specified elsewhere.Other Specified Diabetes Mellitus with Ketoacidosis without Coma|Other specified diabetes mellitus with ketoacidosis without coma
evidence of other specified diabetes mellitus with ketoacidosis without coma not specified elsewhere.Proximal Renal Tubular Acidosis|Type II Renal Tubular Acidosis
impairment in renal proximal tubule bicarbonate reabsorption that results in a hypokalemic hyperchloremic metabolic acidosis, which is most commonly associated with renal fanconi syndrome.Renal Tubular Acidosis Associated With Deafness
renal tubular acidosis associated with sensorineural hearing loss, and which is typically associated with recessive mutations.Renal Tubular Acidosis|renal tubular acidosis
the inability of the kidneys to maintain acid-base homeostasis.Type 1 Diabetes Mellitus with Ketoacidosis with Coma|Type 1 diabetes mellitus with ketoacidosis with coma
evidence of type 1 diabetes mellitus with ketoacidosis with coma.Type 1 Diabetes Mellitus with Ketoacidosis without Coma|Type 1 diabetes mellitus with ketoacidosis without coma
evidence of type 1 diabetes mellitus with ketoacidosis without coma.Type 2 Diabetes Mellitus with Ketoacidosis with Coma|Type 2 diabetes mellitus with ketoacidosis with coma
evidence of type 2 diabetes mellitus with ketoacidosis with coma.Type 2 Diabetes Mellitus with Ketoacidosis without Coma|Type 2 diabetes mellitus with ketoacidosis without coma
evidence of type 2 diabetes mellitus with ketoacidosis without coma.Type I Renal Tubular Acidosis
impairment in renal collecting tubule acid secretion that results in a hypokalemic hyperchloremic metabolic acidosis.
Patient EducationClinical
Common Infant and Newborn Problems
It is hard when your baby is sick. Common health problems in babies include colds, coughs, fevers, and vomiting. Babies also commonly have skin problems, like diaper rash or cradle cap.
Read the full article at MedlinePlus
Courtesy of MedlinePlus, a service of the U.S. National Library of Medicine.
Convert P84 to ICD-9-CMHistory
The closest ICD-9-CM equivalents under the General Equivalence Mappings.
Code HistoryHistory
Questions About P84Overview
Is P84 (Other problems with newborn) a billable code?
Yes. This is a billable ICD-10-CM code, specific enough to report other problems with newborn on HIPAA-covered claims from October 1, 2025 through September 30, 2026.
What is the ICD-9 equivalent of P84?
Under the General Equivalence Mappings, other problems with newborn converts to ICD-9-CM 768.9 (birth asphyxia NOS), 770.88 (NB hypoxia), and 775.81 (NB acidosis NEC). The mapping is approximate, so confirm the match fits the documentation.
Footnotes
[1] Not chronic - A diagnosis code that does not fit the criteria for chronic condition (duration, ongoing medical treatment, and limitations) is considered not chronic. Some codes designated as not chronic are acute conditions. Other diagnosis codes that indicate a possible chronic condition, but for which the duration of the illness is not specified in the code description (i.e., we do not know the condition has lasted 12 months or longer) also are considered not chronic.
