"Myopathy" References in the ICD-10-CM Index to Diseases and Injuries
- Myopathy - G72.9 Myopathy, unspecified
- alcoholic - G72.1 Alcoholic myopathy
- benign congenital - G71.20 Congenital myopathy, unspecifed
- central core - G71.29 Other congenital myopathy
- centronuclear - G71.228 Other centronuclear myopathy
- congenital (benign) - G71.20 Congenital myopathy, unspecifed
- critical illness - G72.81 Critical illness myopathy
- distal - G71.09 Other specified muscular dystrophies
- drug-induced - G72.0 Drug-induced myopathy
- endocrine NEC - E34.9 Endocrine disorder, unspecified
- extraocular muscles - H05.82 Myopathy of extraocular muscles
- facioscapulohumeral - G71.02 Facioscapulohumeral muscular dystrophy
- hereditary - G71.9 Primary disorder of muscle, unspecified
- specified NEC - G71.8 Other primary disorders of muscles
- hyaline body - G71.29 Other congenital myopathy
- immune NEC - G72.49 Other inflammatory and immune myopathies, not elsewhere classified
- in (due to)
- Addison's disease - E27.1 Primary adrenocortical insufficiency
- alcohol - G72.1 Alcoholic myopathy
- amyloidosis - E85.0 Non-neuropathic heredofamilial amyloidosis
- cretinism - E00.9 Congenital iodine-deficiency syndrome, unspecified
- Cushing's syndrome - E24.9 Cushing's syndrome, unspecified
- drugs - G72.0 Drug-induced myopathy
- endocrine disease NEC - E34.9 Endocrine disorder, unspecified
- giant cell arteritis - M31.6 Other giant cell arteritis
- glycogen storage disease - E74.00 Glycogen storage disease, unspecified
- hyperadrenocorticism - E24.9 Cushing's syndrome, unspecified
- hyperparathyroidism NEC - E21.3 Hyperparathyroidism, unspecified
- hypoparathyroidism - E20.9 Hypoparathyroidism, unspecified
- hypopituitarism - E23.0 Hypopituitarism
- hypothyroidism - E03.9 Hypothyroidism, unspecified
- infectious disease NEC - B99 Other and unspecified infectious diseases
- lipid storage disease - E75.6 Lipid storage disorder, unspecified
- metabolic disease NEC - E88.9 Metabolic disorder, unspecified
- myxedema - E03.9 Hypothyroidism, unspecified
- parasitic disease NEC - B89 Unspecified parasitic disease
- polyarteritis nodosa - M30.0 Polyarteritis nodosa
- rheumatoid arthritis - See: Rheumatoid, myopathy;
- sarcoidosis - D86.87 Sarcoid myositis
- scleroderma - M34.82 Systemic sclerosis with myopathy
- sicca syndrome - M35.03 Sicca syndrome with myopathy
- Sjögren's syndrome - M35.03 Sicca syndrome with myopathy
- systemic lupus erythematosus - M32.19 Other organ or system involvement in systemic lupus erythematosus
- thyrotoxicosis (hyperthyroidism) - E05.90 Thyrotoxicosis, unspecified without thyrotoxic crisis or storm
- with thyroid storm - E05.91 Thyrotoxicosis, unspecified with thyrotoxic crisis or storm
- toxic agent NEC - G72.2 Myopathy due to other toxic agents
- inflammatory NEC - G72.49 Other inflammatory and immune myopathies, not elsewhere classified
- intensive care (ICU) - G72.81 Critical illness myopathy
- limb-girdle - G71.09 Other specified muscular dystrophies
- mitochondrial NEC - G71.3 Mitochondrial myopathy, not elsewhere classified
- myosin storage - G71.29 Other congenital myopathy
- myotubular (centronuclear) - G71.220 X-linked myotubular myopathy
- mytonic, proximal (PROMM) - G71.11 Myotonic muscular dystrophy
- nemaline - G71.21 Nemaline myopathy
- ocular - G71.09 Other specified muscular dystrophies
- oculopharyngeal - G71.09 Other specified muscular dystrophies
- of critical illness - G72.81 Critical illness myopathy
- primary - G71.9 Primary disorder of muscle, unspecified
- specified NEC - G71.8 Other primary disorders of muscles
- progressive NEC - G72.89 Other specified myopathies
- proximal myotonic (PROMM) - G71.11 Myotonic muscular dystrophy
- rod (body) - G71.21 Nemaline myopathy
- scapulohumeral - G71.02 Facioscapulohumeral muscular dystrophy
- specified NEC - G72.89 Other specified myopathies
- toxic - G72.2 Myopathy due to other toxic agents
Applicable Clinical Terms Definitions
Addison Disease: An adrenal disease characterized by the progressive destruction of the ADRENAL CORTEX, resulting in insufficient production of ALDOSTERONE and HYDROCORTISONE. Clinical symptoms include ANOREXIA; NAUSEA; WEIGHT LOSS; MUSCLE WEAKNESS; and HYPERPIGMENTATION of the SKIN due to increase in circulating levels of ACTH precursor hormone which stimulates MELANOCYTES.
Alcoholics: Persons who have a history of physical or psychological dependence on ETHANOL.
Amyloidosis: A group of sporadic, familial and/or inherited, degenerative, and infectious disease processes, linked by the common theme of abnormal protein folding and deposition of AMYLOID. As the amyloid deposits enlarge they displace normal tissue structures, causing disruption of function. Various signs and symptoms depend on the location and size of the deposits.
Cushing Syndrome: A condition caused by prolonged exposure to excess levels of cortisol (HYDROCORTISONE) or other GLUCOCORTICOIDS from endogenous or exogenous sources. It is characterized by upper body OBESITY; OSTEOPOROSIS; HYPERTENSION; DIABETES MELLITUS; HIRSUTISM; AMENORRHEA; and excess body fluid. Endogenous Cushing syndrome or spontaneous hypercortisolism is divided into two groups, those due to an excess of ADRENOCORTICOTROPIN and those that are ACTH-independent.
Giant Cell Arteritis: A systemic autoimmune disorder that typically affects medium and large ARTERIES, usually leading to occlusive granulomatous vasculitis with transmural infiltrate containing multinucleated GIANT CELLS. The TEMPORAL ARTERY is commonly involved. This disorder appears primarily in people over the age of 50. Symptoms include FEVER; FATIGUE; HEADACHE; visual impairment; pain in the jaw and tongue; and aggravation of pain by cold temperatures. (From Adams et al., Principles of Neurology, 6th ed)
Glycogen Storage Disease: A group of inherited metabolic disorders involving the enzymes responsible for the synthesis and degradation of glycogen. In some patients, prominent liver involvement is presented. In others, more generalized storage of glycogen occurs, sometimes with prominent cardiac involvement.
Hypoparathyroidism: A condition caused by a deficiency of PARATHYROID HORMONE (or PTH). It is characterized by HYPOCALCEMIA and hyperphosphatemia. Hypocalcemia leads to TETANY. The acquired form is due to removal or injuries to the PARATHYROID GLANDS. The congenital form is due to mutations of genes, such as TBX1; (see DIGEORGE SYNDROME); CASR encoding CALCIUM-SENSING RECEPTOR; or PTH encoding parathyroid hormone.
Hypopituitarism: Diminution or cessation of secretion of one or more hormones from the anterior pituitary gland (including LH; FOLLICLE STIMULATING HORMONE; SOMATOTROPIN; and CORTICOTROPIN). This may result from surgical or radiation ablation, non-secretory PITUITARY NEOPLASMS, metastatic tumors, infarction, PITUITARY APOPLEXY, infiltrative or granulomatous processes, and other conditions.
Hypothyroidism: A syndrome that results from abnormally low secretion of THYROID HORMONES from the THYROID GLAND, leading to a decrease in BASAL METABOLIC RATE. In its most severe form, there is accumulation of MUCOPOLYSACCHARIDES in the SKIN and EDEMA, known as MYXEDEMA. It may be primary or secondary due to other pituitary disease, or hypothalamic dysfunction.
Muscular Diseases: Acquired, familial, and congenital disorders of SKELETAL MUSCLE and SMOOTH MUSCLE.
Myxedema: A condition characterized by a dry, waxy type of swelling (EDEMA) with abnormal deposits of MUCOPOLYSACCHARIDES in the SKIN and other tissues. It is caused by a deficiency of THYROID HORMONES. The skin becomes puffy around the eyes and on the cheeks. The face is dull and expressionless with thickened nose and lips.
Polyarteritis Nodosa: A form of necrotizing non-granulomatous inflammation occurring primarily in medium-sized ARTERIES, often with microaneurysms. It is characterized by muscle, joint, and abdominal pain resulting from arterial infarction and scarring in affected organs. Polyarteritis nodosa with lung involvement is called CHURG-STRAUSS SYNDROME.
Sarcoidosis: An idiopathic systemic inflammatory granulomatous disorder comprised of epithelioid and multinucleated giant cells with little necrosis. It usually invades the lungs with fibrosis and may also involve lymph nodes, skin, liver, spleen, eyes, phalangeal bones, and parotid glands.