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  2. ICD-10-CM Codes List
  3. M00–M99
  4. M80-M85
  5. Adult osteomalacia (M83)

Adult osteomalacia (M83)

M83 codes identify different types of adult osteomalacia, a condition characterized by softening of the bones due to defective bone mineralization. These ICD-10 codes cover causes ranging from nutritional deficiencies to drug-induced and disease-related osteomalacia in adults.

The category includes M83.0 for puerperal osteomalacia, which affects mothers during or after pregnancy. M83.1 defines senile osteomalacia, commonly seen in elderly adults. Codes M83.2 and M83.3 specify osteomalacia caused by malabsorption and malnutrition, respectively, the latter also known as vitamin D deficiency osteomalacia. M83.4 covers aluminum bone disease, linked to aluminum exposure affecting bone health. Drug-related causes are coded at M83.5, including anticonvulsant-induced forms. For other specific adult osteomalacia types, such as those stemming from liver or endocrine diseases or tumors, M83.8 is used. When the exact cause is unclear or unspecified, M83.9 applies. These codes help medical coders accurately classify various presentations and underlying causes of adult osteomalacia.

Instructional Notations

Type 1 Excludes

A type 1 excludes note is a pure excludes note. It means "NOT CODED HERE!" An Excludes1 note indicates that the code excluded should never be used at the same time as the code above the Excludes1 note. An Excludes1 is used when two conditions cannot occur together, such as a congenital form versus an acquired form of the same condition.

  • infantile and juvenile osteomalacia E55.0
  • renal osteodystrophy N25.0
  • rickets active E55.0
  • rickets active sequelae E64.3
  • vitamin D-resistant osteomalacia E83.31
  • vitamin D-resistant rickets active E83.31

Clinical Terms

The following clinical terms provide additional context, helping users better understand the clinical background and common associations for each diagnosis listed in this section. Including related terms alongside ICD-10-CM codes supports coders, billers, and healthcare professionals in improving accuracy, enhancing documentation, and facilitating research or patient education.

Chronic Kidney Disease-Mineral and Bone Disorder

Decalcification of bone or abnormal bone development due to chronic KIDNEY DISEASES, in which 1,25-DIHYDROXYVITAMIN D3 synthesis by the kidneys is impaired, leading to reduced negative feedback on PARATHYROID HORMONE. The resulting SECONDARY HYPERPARATHYROIDISM eventually leads to bone disorders.

Familial Hypophosphatemic Rickets

A hereditary disorder characterized by HYPOPHOSPHATEMIA; RICKETS; OSTEOMALACIA; renal defects in phosphate reabsorption and vitamin D metabolism; and growth retardation. Autosomal and X-linked dominant and recessive variants have been reported.

Osteomalacia

Disorder caused by an interruption of the mineralization of organic bone matrix leading to bone softening, bone pain, and weakness. It is the adult form of rickets resulting from disruption of VITAMIN D; PHOSPHORUS; or CALCIUM homeostasis.

Rickets

Disorders caused by interruption of BONE MINERALIZATION manifesting as OSTEOMALACIA in adults and characteristic deformities in infancy and childhood due to disturbances in normal BONE FORMATION. The mineralization process may be interrupted by disruption of VITAMIN D; PHOSPHORUS; or CALCIUM homeostasis, resulting from dietary deficiencies, or acquired, or inherited metabolic, or hormonal disturbances.

Rickets, Hypophosphatemic

A disorder characterized by HYPOPHOSPHATEMIA; RICKETS; OSTEOMALACIA; resulting from lack of phosphate reabsorption by the kidneys and possible defects in vitamin D metabolism.