Vitamin D deficiency (E55)
The ICD-10 code section E55 is used to identify and classify different forms of vitamin D deficiency, including specific conditions like active rickets and unspecified vitamin D deficiency. These codes help accurately capture health issues related to inadequate vitamin D levels that affect bone health and metabolism.
E55.0 is designated for active rickets, a condition resulting from vitamin D deficiency that leads to bone deformities, such as curvature of the spine, hypophosphatasia rickets, and osteomalacia. Common synonyms like "rachitic dwarf," "rachitic rosary," and "hypocalcemic rickets" refer to this same bone disorder caused by insufficient vitamin D. Meanwhile, E55.9 covers the broader category of vitamin D deficiency where the cause or specifics are not clearly defined. This includes vitamin D deficiency linked to chronic kidney disease, secondary hyperparathyroidism, or inadequate vitamin D intake. Using the ICD-10 code for vitamin D deficiency ensures proper documentation for nutritional deficiencies impacting bone and endocrine health.
Endocrine, nutritional and metabolic diseases (E00–E89)
Other nutritional deficiencies (E50-E64)
E55 Vitamin D deficiency
- E55.0 Rickets, active
- E55.9 Vitamin D deficiency, unspecified
Vitamin D deficiency (E55)
Instructional Notations
Type 1 Excludes
A type 1 excludes note is a pure excludes note. It means "NOT CODED HERE!" An Excludes1 note indicates that the code excluded should never be used at the same time as the code above the Excludes1 note. An Excludes1 is used when two conditions cannot occur together, such as a congenital form versus an acquired form of the same condition.
Clinical Terms
The following clinical terms provide additional context, helping users better understand the clinical background and common associations for each diagnosis listed in this section. Including related terms alongside ICD-10-CM codes supports coders, billers, and healthcare professionals in improving accuracy, enhancing documentation, and facilitating research or patient education.
Chronic Kidney Disease-Mineral and Bone Disorder
Decalcification of bone or abnormal bone development due to chronic KIDNEY DISEASES, in which 1,25-DIHYDROXYVITAMIN D3 synthesis by the kidneys is impaired, leading to reduced negative feedback on PARATHYROID HORMONE. The resulting SECONDARY HYPERPARATHYROIDISM eventually leads to bone disorders.
Familial Hypophosphatemic Rickets
A hereditary disorder characterized by HYPOPHOSPHATEMIA; RICKETS; OSTEOMALACIA; renal defects in phosphate reabsorption and vitamin D metabolism; and growth retardation. Autosomal and X-linked dominant and recessive variants have been reported.
Osteomalacia
Disorder caused by an interruption of the mineralization of organic bone matrix leading to bone softening, bone pain, and weakness. It is the adult form of rickets resulting from disruption of VITAMIN D; PHOSPHORUS; or CALCIUM homeostasis.
Rickets
Disorders caused by interruption of BONE MINERALIZATION manifesting as OSTEOMALACIA in adults and characteristic deformities in infancy and childhood due to disturbances in normal BONE FORMATION. The mineralization process may be interrupted by disruption of VITAMIN D; PHOSPHORUS; or CALCIUM homeostasis, resulting from dietary deficiencies, or acquired, or inherited metabolic, or hormonal disturbances.
Rickets, Hypophosphatemic
A disorder characterized by HYPOPHOSPHATEMIA; RICKETS; OSTEOMALACIA; resulting from lack of phosphate reabsorption by the kidneys and possible defects in vitamin D metabolism.
Vitamin D Deficiency
A nutritional condition produced by a deficiency of VITAMIN D in the diet, insufficient production of vitamin D in the skin, inadequate absorption of vitamin D from the diet, or abnormal conversion of vitamin D to its bioactive metabolites. It is manifested clinically as RICKETS in children and OSTEOMALACIA in adults. (From Cecil Textbook of Medicine, 19th ed, p1406)
