ICD-10 Diagnosis Code N07.5

Hereditary nephropathy, NEC w diffuse mesangiocap glomrlneph

Diagnosis Code N07.5

ICD-10: N07.5
Short Description: Hereditary nephropathy, NEC w diffuse mesangiocap glomrlneph
Long Description: Hereditary nephropathy, not elsewhere classified with diffuse mesangiocapillary glomerulonephritis
This is the 2018 version of the ICD-10-CM diagnosis code N07.5

Valid for Submission
The code N07.5 is valid for submission for HIPAA-covered transactions.

Code Classification
  • Diseases of the genitourinary system (N00–N99)
    • Glomerular diseases (N00-N08)
      • Hereditary nephropathy, not elsewhere classified (N07)

Information for Medical Professionals

Diagnostic Related Groups
The diagnosis code N07.5 is grouped in the following Diagnostic Related Group(s) (MS-DRG V35.0)


Convert to ICD-9 Additional informationCallout TooltipGeneral Equivalence Map
The ICD-10 and ICD-9 GEMs are used to facilitate linking between the diagnosis codes in ICD-9-CM and the new ICD-10-CM code set. The GEMs are the raw material from which providers, health information vendors and payers can derive specific applied mappings to meet their needs.

  • Familial lobular glomerulopathy
  • Hereditary diffuse mesangial proliferative glomerulonephritis
  • Hereditary diffuse mesangiocapillary glomerulonephritis
  • Hereditary mesangiocapillary glomerulonephritis, type 2
  • Hereditary nephropathy co-occurrent with membranoproliferative glomerulonephritis type III
  • Mesangiocapillary glomerulonephritis type III
  • Mesangiocapillary glomerulonephritis, type II

Index of Diseases and Injuries
References found for the code N07.5 in the Index of Diseases and Injuries:

Information for Patients

Genetic Disorders

Genes are the building blocks of heredity. They are passed from parent to child. They hold DNA, the instructions for making proteins. Proteins do most of the work in cells. They move molecules from one place to another, build structures, break down toxins, and do many other maintenance jobs.

Sometimes there is a mutation, a change in a gene or genes. The mutation changes the gene's instructions for making a protein, so the protein does not work properly or is missing entirely. This can cause a medical condition called a genetic disorder.

You can inherit a gene mutation from one or both parents. A mutation can also happen during your lifetime.

There are three types of genetic disorders:

  • Single-gene disorders, where a mutation affects one gene. Sickle cell anemia is an example.
  • Chromosomal disorders, where chromosomes (or parts of chromosomes) are missing or changed. Chromosomes are the structures that hold our genes. Down syndrome is a chromosomal disorder.
  • Complex disorders, where there are mutations in two or more genes. Often your lifestyle and environment also play a role. Colon cancer is an example.

Genetic tests on blood and other tissue can identify genetic disorders.

NIH: National Library of Medicine

  • Genetics (Medical Encyclopedia)

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Kidney Diseases

Also called: Renal disease

You have two kidneys, each about the size of your fist. They are near the middle of your back, just below the rib cage. Inside each kidney there are about a million tiny structures called nephrons. They filter your blood. They remove wastes and extra water, which become urine. The urine flows through tubes called ureters. It goes to your bladder, which stores the urine until you go to the bathroom.

Most kidney diseases attack the nephrons. This damage may leave kidneys unable to remove wastes. Causes can include genetic problems, injuries, or medicines. You have a higher risk of kidney disease if you have diabetes, high blood pressure, or a close family member with kidney disease. Chronic kidney disease damages the nephrons slowly over several years. Other kidney problems include

  • Cancer
  • Cysts
  • Stones
  • Infections

Your doctor can do blood and urine tests to check if you have kidney disease. If your kidneys fail, you will need dialysis or a kidney transplant.

NIH: National Institute of Diabetes and Digestive and Kidney Diseases

  • ACE inhibitors (Medical Encyclopedia)
  • Acute nephritic syndrome (Medical Encyclopedia)
  • Analgesic nephropathy (Medical Encyclopedia)
  • Atheroembolic renal disease (Medical Encyclopedia)
  • Bartter syndrome (Medical Encyclopedia)
  • Bilateral hydronephrosis (Medical Encyclopedia)
  • Congenital nephrotic syndrome (Medical Encyclopedia)
  • Distal renal tubular acidosis (Medical Encyclopedia)
  • Focal segmental glomerulosclerosis (Medical Encyclopedia)
  • Glomerulonephritis (Medical Encyclopedia)
  • Goodpasture syndrome (Medical Encyclopedia)
  • IgA nephropathy (Medical Encyclopedia)
  • Injury - kidney and ureter (Medical Encyclopedia)
  • Interstitial nephritis (Medical Encyclopedia)
  • Kidney removal (Medical Encyclopedia)
  • Kidney removal - discharge (Medical Encyclopedia)
  • Medicines and Kidney Disease - NIH (National Kidney Disease Education Program)
  • Membranoproliferative GN I (Medical Encyclopedia)
  • Membranous nephropathy (Medical Encyclopedia)
  • Minimal change disease (Medical Encyclopedia)
  • Nephrocalcinosis (Medical Encyclopedia)
  • Nephrotic syndrome (Medical Encyclopedia)
  • Obstructive uropathy (Medical Encyclopedia)
  • Perirenal abscess (Medical Encyclopedia)
  • Proximal renal tubular acidosis (Medical Encyclopedia)
  • Reflux nephropathy (Medical Encyclopedia)
  • Renal papillary necrosis (Medical Encyclopedia)
  • Renal perfusion scintiscan (Medical Encyclopedia)
  • Renal vein thrombosis (Medical Encyclopedia)
  • Unilateral hydronephrosis (Medical Encyclopedia)

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Fibronectin glomerulopathy Fibronectin glomerulopathy is a kidney disease that usually develops between early and mid-adulthood but can occur at any age. It eventually leads to irreversible kidney failure (end-stage renal disease).Individuals with fibronectin glomerulopathy usually have blood and excess protein in their urine (hematuria and proteinuria, respectively). They also have high blood pressure (hypertension). Some affected individuals develop renal tubular acidosis, which occurs when the kidneys are unable to remove enough acid from the body and the blood becomes too acidic.The kidneys of people with fibronectin glomerulopathy have large deposits of the protein fibronectin-1 in structures called glomeruli. These structures are clusters of tiny blood vessels in the kidneys that filter waste products from blood. The waste products are then released in urine. The fibronectin-1 deposits impair the glomeruli's filtration ability.Fifteen to 20 years following the appearance of signs and symptoms, individuals with fibronectin glomerulopathy often develop end-stage renal disease. Affected individuals may receive treatment in the form of a kidney transplant; in some cases, fibronectin glomerulopathy comes back (recurs) following transplantation.
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