2024 ICD-10-CM Diagnosis Code R47.01

Approximate Synonyms

The following clinical terms are approximate synonyms or lay terms that might be used to identify the correct diagnosis code:

• Anomic aphasia
• Aphasia
• Aphasia due to brain damage
• Aphasia, agnosia, dyslexia AND/OR apraxia
• Associative aphasia
• Atypical progressive supranuclear palsy syndrome
• Combined aphasia
• Commissural aphasia
• Complete aphasia
• Conduction aphasia
• Functional aphasia
• Gibberish aphasia
• Global aphasia
• Graphomotor aphasia
• Intellectual aphasia
• Language disorder associated with thought disorder
• Motor aphasia
• Mutism
• Paragrammatism
• Parieto-occipital aphasia
• Pathematic aphasia
• Posttraumatic aphasia
• Post-traumatic mutism
• Progressive supranuclear palsy
• Progressive supranuclear palsy progressive non fluent aphasia
• Semantic aphasia
• Subcortical aphasia
• Supranuclear gaze palsy
• Supranuclear paralysis
• Syntactical aphasia
• Temporal lobe aphasia
• Transcortical aphasia

Clinical Information

• Anomia

  a language dysfunction characterized by the inability to name people and objects that are correctly perceived. the individual is able to describe the object in question, but cannot provide the name. this condition is associated with lesions of the dominant hemisphere involving the language areas, in particular the temporal lobe. (from adams et al., principles of neurology, 6th ed, p484)

• Aphasia

  a cognitive disorder marked by an impaired ability to comprehend or express language in its written or spoken form. this condition is caused by diseases which affect the language areas of the dominant hemisphere. clinical features are used to classify the various subtypes of this condition. general categories include receptive, expressive, and mixed forms of aphasia.

• Aphasia, Broca

  an aphasia characterized by impairment of expressive language (speech, writing, signs) and relative preservation of receptive language abilities (i.e., comprehension). this condition is caused by lesions of the motor association cortex in the frontal lobe (broca area and adjacent cortical and white matter regions).

• Aphasia, Conduction

  a type of fluent aphasia characterized by an impaired ability to repeat one and two word phrases, despite retained comprehension. this condition is associated with dominant hemisphere lesions involving the arcuate fasciculus (a white matter projection between broca's and wernicke's areas) and adjacent structures. like patients with wernicke aphasia (aphasia, wernicke), patients with conduction aphasia are fluent but commit paraphasic errors during attempts at written and oral forms of communication. (from adams et al., principles of neurology, 6th ed, p482; brain & bannister, clinical neurology, 7th ed, p142; kandel et al., principles of neural science, 3d ed, p848)

• Aphasia, Primary Progressive

  a progressive form of dementia characterized by the global loss of language abilities and initial preservation of other cognitive functions. fluent and nonfluent subtypes have been described. eventually a pattern of global cognitive dysfunction, similar to alzheimer disease, emerges. pathologically, there are no alzheimer or pick disease like changes, however, spongiform changes of cortical layers ii and iii are present in the temporal lobe and frontal lobe. (from brain 1998 jan;121(pt 1):115-26)

• Aphasia, Wernicke

  impairment in the comprehension of speech and meaning of words, both spoken and written, and of the meanings conveyed by their grammatical relationships in sentences. it is caused by lesions that primarily affect wernicke's area, which lies in the posterior perisylvian region of the temporal lobe of the dominant hemisphere. (from brain & bannister, clinical neurology, 7th ed, p141; kandel et al., principles of neural science, 3d ed, p846)

• Landau-Kleffner Syndrome

  a syndrome characterized by the onset of isolated language dysfunction in otherwise normal children (age of onset 4-7 years) and epileptiform discharges on electroencephalography. seizures, including atypical absence (epilepsy, absence), complex partial (epilepsy, complex partial), and other types may occur. the electroencephalographic abnormalities and seizures tend to resolve by puberty. the language disorder may also resolve although some individuals are left with severe language dysfunction, including aphasia and auditory agnosia. (from menkes, textbook of child neurology, 5th ed, pp749-50; j child neurol 1997 nov;12(8):489-495).

• Mutism

  the inability to generate oral-verbal expression, despite normal comprehension of speech. this may be associated with brain diseases or mental disorders. organic mutism may be associated with damage to the frontal lobe; brain stem; thalamus; and cerebellum. selective mutism is a psychological condition that usually affects children characterized by continuous refusal to speak in social situations by a child who is able and willing to speak to selected persons. kussmal aphasia refers to mutism in psychosis. (from fortschr neurol psychiatr 1994; 62(9):337-44)

• Neuropsychological Tests

  tests designed to assess neurological function associated with certain behaviors. they are used in diagnosing brain dysfunction or damage and central nervous system disorders or injury.

• Primary Progressive Nonfluent Aphasia

  a form of frontotemporal lobar degeneration and a progressive form of dementia characterized by motor speech impairment and agrammatism, with relative sparing of single word comprehension and semantic memory.

• Akinetic Mutism

  a syndrome characterized by a silent and inert state without voluntary motor activity despite preserved sensorimotor pathways and vigilance. bilateral frontal lobe dysfunction involving the anterior cingulate gyrus and related brain injuries are associated with this condition. this may result in impaired abilities to communicate and initiate motor activities. (from adams et al., principles of neurology, 6th ed, p348; fortschr neurol psychiatr 1995 feb;63(2):59-67)

• Pseudobulbar Palsy

  a syndrome characterized by dysarthria, dysphagia, dysphonia, impairment of voluntary movements of tongue and facial muscles, and emotional lability. this condition is caused by diseases that affect the motor fibers that travel from the cerebral cortex to the lower brain stem (i.e., corticobulbar tracts); including multiple sclerosis; motor neuron disease; and cerebrovascular disorders. (from adams et al., principles of neurology, 6th ed, p489)

• Frontal Lobe

  the part of the cerebral hemisphere anterior to the central sulcus, and anterior and superior to the lateral sulcus.

• Temporal Lobe

  lower lateral part of the cerebral hemisphere responsible for auditory, olfactory, and semantic processing. it is located inferior to the lateral fissure and anterior to the occipital lobe.

• Anomia|Nominal Aphasia

  impaired ability to retrieve words; in particular, an inability to recall the names of objects and people.

• Aphasia

  impairment of the expression or comprehension of verbal and written language.

• Aphasia following Cerebral Infarction|Aphasia following cerebral infarction

  evidence of aphasia following cerebral infarction.

• Aphasia following Other Cerebrovascular Disease|Aphasia following other cerebrovascular disease

  evidence of aphasia following other cerebrovascular disease.

• Aphasia following Unspecified Cerebrovascular Disease|Aphasia following unspecified cerebrovascular disease

  evidence of aphasia following unspecified cerebrovascular disease.

• Broca's Aphasia|Motor Aphasia

  aphasia characterized by impairment of expressive language due to injury to the motor association cortex in the frontal lobe.

• Epilepsy, Focal, with Speech Disorder and with or without Mental Retardation|Aphasia, Acquired, with Epilepsy|FESD with or without Mental Retardation

  an autosomal dominant condition caused by mutation(s) in the grin2a gene, encoding glutamate receptor ionotropic nmda 2a. it is characterized by childhood-onset seizures affecting the temporal lobe or rolandic area of the brain, with a variable phenotype. it often affects speech and language.

• Landau-Kleffner Syndrome|Acquired Epileptic Aphasia

  a rare childhood syndrome characterized by the progressive or sudden inability to understand and use spoken language (aphasia) and paroxysmal electrical brain waves. patients develop epileptic seizures and behavioral changes.

• MASA Syndrome|CRASH Syndrome|Gareis-Mason Syndrome|Mental retardation, Aphasia, Shuffling Gait, and Adducted Thumbs|SPG1|Spastic Paraplegia, X-linked

  an x-linked recessive condition caused by mutation(s) in the l1cam gene, encoding neural cell adhesion molecule l1. it is characterized by mental retardation, aphasia, shuffling gait and adducted thumbs.

• Primary Progressive Aphasia

  a rare neurologic progressive disorder characterized by impairment of the language and speech abilities.

• Primary Progressive Nonfluent Aphasia

  a rare neurologic progressive disorder characterized by impairment of the production of speech.

Convert R47.01 to ICD-9-CM

• ICD-9-CM Code: 784.3 - Aphasia

Code History

• FY 2024 - No Change, effective from 10/1/2023 through 9/30/2024
• FY 2023 - No Change, effective from 10/1/2022 through 9/30/2023
• FY 2022 - No Change, effective from 10/1/2021 through 9/30/2022
• FY 2021 - No Change, effective from 10/1/2020 through 9/30/2021
• FY 2020 - No Change, effective from 10/1/2019 through 9/30/2020
• FY 2019 - No Change, effective from 10/1/2018 through 9/30/2019
• FY 2018 - No Change, effective from 10/1/2017 through 9/30/2018
• FY 2017 - No Change, effective from 10/1/2016 through 9/30/2017
• FY 2016 - New Code, effective from 10/1/2015 through 9/30/2016. This was the first year ICD-10-CM was implemented into the HIPAA code set.