2024 ICD-10-CM Diagnosis Code T45.2X2A

Poisoning by vitamins, intentional self-harm, initial encounter

ICD-10-CM Code:
T45.2X2A
ICD-10 Code for:
Poisoning by vitamins, intentional self-harm, init encntr
Is Billable?
Yes - Valid for Submission
Chronic Condition Indicator: [1]
Not chronic
Code Navigator:

Code Classification

  • Injury, poisoning and certain other consequences of external causes
    (S00–T88)
    • Poisoning by, adverse effect of and underdosing of drugs, medicaments and biological substances
      (T36-T50)
      • Poisoning by, adverse effect of and underdosing of primarily systemic and hematological agents, not elsewhere classified
        (T45)

T45.2X2A is a billable diagnosis code used to specify a medical diagnosis of poisoning by vitamins, intentional self-harm, initial encounter. The code is valid during the current fiscal year for the submission of HIPAA-covered transactions from October 01, 2023 through September 30, 2024.

T45.2X2A is an initial encounter code, includes a 7th character and should be used while the patient is receiving active treatment for a condition like poisoning by vitamins intentional self-harm. According to ICD-10-CM Guidelines an "initial encounter" doesn't necessarily means "initial visit". The 7th character should be used when the patient is undergoing active treatment regardless if new or different providers saw the patient over the course of a treatment. The appropriate 7th character codes should also be used even if the patient delayed seeking treatment for a condition.

Approximate Synonyms

The following clinical terms are approximate synonyms or lay terms that might be used to identify the correct diagnosis code:

  • Intentional vitamin A overdose
  • Intentional vitamin A poisoning
  • Intentional vitamin D and/or vitamin D derivative overdose
  • Intentional vitamin D poisoning
  • Poisoning by vitamin A
  • Poisoning by vitamin A
  • Poisoning by vitamin D
  • Poisoning by vitamin D
  • Vitamin A overdose
  • Vitamin D and/or vitamin D derivative overdose

Clinical Classification

Clinical CategoryCCSR Category CodeInpatient Default CCSROutpatient Default CCSR
External cause codes: intent of injury, self-harmEXT021N - Not default inpatient assignment for principal diagnosis or first-listed diagnosis.N - Not default outpatient assignment for principal diagnosis or first-listed diagnosis.
External cause codes: poisoning by drugEXT014N - Not default inpatient assignment for principal diagnosis or first-listed diagnosis.N - Not default outpatient assignment for principal diagnosis or first-listed diagnosis.
Poisoning by drugs, initial encounterINJ022N - Not default inpatient assignment for principal diagnosis or first-listed diagnosis.N - Not default outpatient assignment for principal diagnosis or first-listed diagnosis.
Suicidal ideation/attempt/intentional self-harmMBD012Y - Yes, default inpatient assignment for principal diagnosis or first-listed diagnosis.Y - Yes, default outpatient assignment for principal diagnosis or first-listed diagnosis.

Clinical Information

  • Adenine

    a purine base and a fundamental unit of adenine nucleotides.
  • Adenine Nucleotide Translocator 1

    a subtype of mitochondrial adp, atp translocase found primarily in heart muscle (myocardium) and skeletal muscle (muscle, skeletal).
  • Adenine Nucleotide Translocator 2

    a subtype of mitochondrial adp, atp translocase found primarily in fibroblasts.
  • Adenine Nucleotide Translocator 3

    a subtype of mitochondrial adp, atp translocase found primarily in the liver.
  • Adenine Nucleotides

  • Adenine Phosphoribosyltransferase

    an enzyme catalyzing the formation of amp from adenine and phosphoribosylpyrophosphate. it can act as a salvage enzyme for recycling of adenine into nucleic acids. ec 2.4.2.7.
  • Deoxyadenosines

    adenosine molecules which can be substituted in any position, but are lacking one hydroxyl group in the ribose part of the molecule.
  • Mitochondrial ADP, ATP Translocases

    a class of nucleotide translocases found abundantly in mitochondria that function as integral components of the inner mitochondrial membrane. they facilitate the exchange of adp and atp between the cytosol and the mitochondria, thereby linking the subcellular compartments of atp production to those of atp utilization.
  • NAD

    a coenzyme composed of ribosylnicotinamide 5'-diphosphate coupled to adenosine 5'-phosphate by pyrophosphate linkage. it is found widely in nature and is involved in numerous enzymatic reactions in which it serves as an electron carrier by being alternately oxidized (nad+) and reduced (nadh). (dorland, 27th ed)
  • Poly A

    a group of adenine ribonucleotides in which the phosphate residues of each adenine ribonucleotide act as bridges in forming diester linkages between the ribose moieties.
  • Vidarabine

    a nucleoside antibiotic isolated from streptomyces antibioticus. it has some antineoplastic properties and has broad spectrum activity against dna viruses in cell cultures and significant antiviral activity against infections caused by a variety of viruses such as the herpes viruses, the vaccinia virus and varicella zoster virus.
  • Vidarabine Phosphate

    an adenosine monophosphate analog in which ribose is replaced by an arabinose moiety. it is the monophosphate ester of vidarabine with antiviral and possibly antineoplastic properties.
  • Ascorbic Acid

    a six carbon compound related to glucose. it is found naturally in citrus fruits and many vegetables. ascorbic acid is an essential nutrient in human diets, and necessary to maintain connective tissue and bone. its biologically active form, vitamin c, functions as a reducing agent and coenzyme in several metabolic pathways. vitamin c is considered an antioxidant.
  • Ascorbic Acid Deficiency

    a condition due to a dietary deficiency of ascorbic acid (vitamin c), characterized by malaise, lethargy, and weakness. as the disease progresses, joints, muscles, and subcutaneous tissues may become the sites of hemorrhage. ascorbic acid deficiency frequently develops into scurvy in young children fed unsupplemented cow's milk exclusively during their first year. it develops also commonly in chronic alcoholism. (cecil textbook of medicine, 19th ed, p1177)
  • Biotin

    a water-soluble, enzyme co-factor present in minute amounts in every living cell. it occurs mainly bound to proteins or polypeptides and is abundant in liver, kidney, pancreas, yeast, and milk.
  • Biotinidase

    an enzyme which catalyzes the release of biotin from biocytin. in human, defects in the enzyme are the cause of the organic acidemia multiple carboxylase deficiency or biotinidase deficiency.
  • Biotinidase Deficiency

    the late onset form of multiple carboxylase deficiency (deficiency of the activities of biotin-dependent enzymes propionyl-coa carboxylase, methylcrotonyl-coa carboxylase, and pyruvate carboxylase) due to a defect or deficiency in biotinidase which is essential for recycling biotin.
  • Biotinylation

    incorporation of biotinyl groups into molecules.
  • Myoclonic Epilepsies, Progressive

    a heterogeneous group of primarily familial epilepsy disorders characterized by myoclonic seizures, tonic-clonic seizures, ataxia, progressive intellectual deterioration, and neuronal degeneration. these include lafora disease; merrf syndrome; neuronal ceroid-lipofuscinosis; sialidosis (see mucolipidoses), and unverricht-lundborg syndrome.
  • Calcifediol

    the major circulating metabolite of vitamin d3. it is produced in the liver and is the best indicator of the body's vitamin d stores. it is effective in the treatment of rickets and osteomalacia, both in azotemic and non-azotemic patients. calcifediol also has mineralizing properties.
  • Calcitriol

    the physiologically active form of vitamin d. it is formed primarily in the kidney by enzymatic hydroxylation of 25-hydroxycholecalciferol (calcifediol). its production is stimulated by low blood calcium levels and parathyroid hormone. calcitriol increases intestinal absorption of calcium and phosphorus, and in concert with parathyroid hormone increases bone resorption.
  • Receptors, Calcitriol

    proteins, usually found in the cytoplasm, that specifically bind calcitriol, migrate to the nucleus, and regulate transcription of specific segments of dna with the participation of d receptor interacting proteins (called drip). vitamin d is converted in the liver and kidney to calcitriol and ultimately acts through these receptors.
  • Vitamin D3 24-Hydroxylase

    a cytochrome p-450 enzyme that has specificity for cholecalciferol (vitamin d3). it hydroxylates the molecule at carbon position 24.
  • Cholecalciferol

    derivative of 7-dehydroxycholesterol formed by ultraviolet rays breaking of the c9-c10 bond. it differs from ergocalciferol in having a single bond between c22 and c23 and lacking a methyl group at c24.
  • Dihydrotachysterol

    a vitamin d that can be regarded as a reduction product of vitamin d2.
  • Esculin

    a derivative of coumarin with molecular formula c15h16o9.
  • Niacinamide

    an important compound functioning as a component of the coenzyme nad. its primary significance is in the prevention and/or cure of blacktongue and pellagra. most animals cannot manufacture this compound in amounts sufficient to prevent nutritional deficiency and it therefore must be supplemented through dietary intake.
  • Pantothenic Acid

    a butyryl-beta-alanine that can also be viewed as pantoic acid complexed with beta alanine. it is incorporated into coenzyme a and protects cells against peroxidative damage by increasing the level of glutathione.
  • Pyridoxal Phosphate

    this is the active form of vitamin b 6 serving as a coenzyme for synthesis of amino acids, neurotransmitters (serotonin, norepinephrine), sphingolipids, aminolevulinic acid. during transamination of amino acids, pyridoxal phosphate is transiently converted into pyridoxamine phosphate (pyridoxamine).
  • Pyridoxal Kinase

    an enzyme that catalyzes reversibly the phosphorylation of pyridoxal in the presence of atp with the formation of pyridoxal 5-phosphate and adp. pyridoxine, pyridoxamine and various derivatives can also act as acceptors. ec 2.7.1.35.
  • Pyridoxaminephosphate Oxidase

    an enzyme catalyzing the deamination of pyridoxaminephosphate to pyridoxal phosphate. it is a flavoprotein that also oxidizes pyridoxine-5-phosphate and pyridoxine. ec 1.4.3.5.
  • Pyridoxic Acid

    the catabolic product of most of vitamin b 6; (pyridoxine; pyridoxal; and pyridoxamine) which is excreted in the urine.
  • Pyridoxine

    the 4-methanol form of vitamin b 6 which is converted to pyridoxal phosphate which is a coenzyme for synthesis of amino acids, neurotransmitters (serotonin, norepinephrine), sphingolipids, aminolevulinic acid. although pyridoxine and vitamin b 6 are still frequently used as synonyms, especially by medical researchers, this practice is erroneous and sometimes misleading (ee snell; ann ny acad sci, vol 585 pg 1, 1990).
  • Pyrithioxin

    a neurotropic agent which reduces permeability of blood-brain barrier to phosphate. it has no vitamin b6 activity.
  • Vitamin B 6 Deficiency

    a nutritional condition produced by a deficiency of vitamin b 6 in the diet, characterized by dermatitis, glossitis, cheilosis, and stomatitis. marked deficiency causes irritability, weakness, depression, dizziness, peripheral neuropathy, and seizures. in infants and children typical manifestations are diarrhea, anemia, and seizures. deficiency can be caused by certain medications, such as isoniazid.
  • Flavin Mononucleotide

    a coenzyme for a number of oxidative enzymes including nadh dehydrogenase. it is the principal form in which riboflavin is found in cells and tissues.
  • Riboflavin

    nutritional factor found in milk, eggs, malted barley, liver, kidney, heart, and leafy vegetables. the richest natural source is yeast. it occurs in the free form only in the retina of the eye, in whey, and in urine; its principal forms in tissues and cells are as flavin mononucleotide and flavin-adenine dinucleotide.
  • Riboflavin Deficiency

    a dietary deficiency of riboflavin causing a syndrome chiefly marked by cheilitis, angular stomatitis, glossitis associated with a purplish red or magenta-colored tongue that may show fissures, corneal vascularization, dyssebacia, and anemia. (dorland, 27th ed)
  • Riboflavin Synthase

    an enzyme that catalyzes the formation of riboflavin from two molecules of 6,7-dimethyl-8-ribityllumazine, utilizing a four-carbon fragment from one molecule which is transferred to the second molecule. ec 2.5.1.9.
  • Fursultiamin

    compound used for therapy of thiamine deficiency. it has also been suggested for several non-deficiency disorders but has not yet proven useful.
  • Maple Syrup Urine Disease

    an autosomal recessive inherited disorder with multiple forms of phenotypic expression, caused by a defect in the oxidative decarboxylation of branched-chain amino acids (amino acids, branched-chain). these metabolites accumulate in body fluids and render a "maple syrup" odor. the disease is divided into classic, intermediate, intermittent, and thiamine responsive subtypes. the classic form presents in the first week of life with ketoacidosis, hypoglycemia, emesis, neonatal seizures, and hypertonia. the intermediate and intermittent forms present in childhood or later with acute episodes of ataxia and vomiting. (from adams et al., principles of neurology, 6th ed, p936)
  • Thiamin Pyrophosphokinase

    an enzyme that catalyzes the formation of thiamine pyrophosphate from atp and thiamine. ec 2.7.6.2.
  • Thiamine

    3-((4-amino-2-methyl-5-pyrimidinyl)methyl)-5-(2- hydroxyethyl)-4-methylthiazolium chloride.
  • Thiamine Deficiency

    a nutritional condition produced by a deficiency of thiamine in the diet, characterized by anorexia, irritability, and weight loss. later, patients experience weakness, peripheral neuropathy, headache, and tachycardia. in addition to being caused by a poor diet, thiamine deficiency in the united states most commonly occurs as a result of alcoholism, since ethanol interferes with thiamine absorption. in countries relying on polished rice as a dietary staple, beriberi prevalence is very high. (from cecil textbook of medicine, 19th ed, p1171)
  • Thiamine Monophosphate

    thiamine dihydrogen phosphate ester. the monophosphate ester of thiamine. synonyms: monophosphothiamine; vitamin b1 monophosphate.
  • Thiamine Pyrophosphatase

    an enzyme that hydrolyzes thiamine pyrophosphate to thiamine monophosphate plus inorganic phosphate. ec 3.6.1.-.
  • Thiamine Pyrophosphate

    the coenzyme form of vitamin b1 present in many animal tissues. it is a required intermediate in the pyruvate dehydrogenase complex and the ketoglutarate dehydrogenase complex.
  • Thiamine Triphosphate

    3-((4-amino-2-methyl-5-pyrimidinyl)methyl)-4-methyl-5-(4,6,8,8-tetrahydroxy-3,5,7-trioxa-4,6,8-triphosphaoct-1-yl)thiazolium hydroxide, inner salt, p,p',p''-trioxide. the triphosphate ester of thiamine. in leigh's disease, this compound is present in decreased amounts in the brain due to a metabolic block in its formation.
  • Thiamin-Triphosphatase

    an enzyme present in nerve tissue. it catalyzes reversibly the formation of thiamine diphosphate and orthophosphate from thiamine triphosphate. ec 3.6.1.28.

Coding Guidelines

When coding a poisoning or reaction to the improper use of a medication (e.g., overdose, wrong substance given or taken in error, wrong route of administration), first assign the appropriate code from categories T36-T50. The poisoning codes have an associated intent as their 5th or 6th character (accidental, intentional self-harm, assault and undetermined. If the intent of the poisoning is unknown or unspecified, code the intent as accidental intent. The undetermined intent is only for use if the documentation in the record specifies that the intent cannot be determined. Use additional code(s) for all manifestations of poisonings.

The appropriate 7th character is to be added to each code from block Poisoning by, adverse effect of and underdosing of primarily systemic and hematological agents, not elsewhere classified (T45). Use the following options for the aplicable episode of care:

  • A - initial encounter
  • D - subsequent encounter
  • S - sequela

Convert T45.2X2A to ICD-9-CM

  • ICD-9-CM Code: 963.5 - Poisoning-vitamins NEC
    Combination Flag - Multiple codes are needed to describe the source diagnosis code. Correct coding should be done based on contextual judgment.
  • ICD-9-CM Code: E950.4 - Poison-drug/medicin NEC
    Combination Flag - Multiple codes are needed to describe the source diagnosis code. Correct coding should be done based on contextual judgment.

Table of Drugs and Chemicals

The parent code T45.2X2 of the current diagnosis code is referenced in the Table of Drugs and Chemicals, this table contains a classification of drugs, industrial solvents, corrosive gases, noxious plants, pesticides, and other toxic agents.

According to ICD-10-CM coding guidelines it is advised to do not code directly from the Table of Drugs and Chemicals, instead always refer back to the Tabular List when doing the initial coding. Each substance in the table is assigned a code according to the poisoning classification and external causes of adverse effects. It is important to use as many codes as necessary to specify all reported drugs, medicinal or chemical substances. If the same diagnosis code describes the causative agent for more than one adverse reaction, poisoning, toxic effect or underdosing, utilize the code only once.

Substance Poisoning
Accidental
(unintentional)
Poisoning
Accidental
(self-harm)
Poisoning
Assault
Poisoning
Undetermined
Adverse
effect
Underdosing
AcetiamineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
AdenineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Adenine
  »arabinoside
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
AlfacalcidolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
AneurineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Ascorbic acidT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
AxerophtholT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
BenfotiamineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
BetacaroteneT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
BiotinT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
BisbentiamineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
BisbutiamineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
CalcifediolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
CalciferolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
CalcitriolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
CaroteneT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
CetotiamineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Cevitamic acidT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
CholecalciferolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
CobalamineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Cod-liver oilT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
ColecalciferolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
CozymeT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
DexpanthenolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Dextro calcium pantothenateT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Dextro pantothenyl alcoholT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
DihydrotachysterolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
ErgocalciferolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
EsculinT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
EsculosideT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Flavine adenine dinucleotideT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
FursultiamineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
IlopanT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
LactoflavinT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
NiacinamideT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
NicotinamideT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
OctotiamineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Oleovitamin AT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Pangamic acidT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
PanthenolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Panthenol
  »topical
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Pantothenic acidT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
ProsultiamineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Provitamin AT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Pyridoxal phosphateT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
PyridoxineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
RetinolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
RiboflavinT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
ThiamineT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
TocoferolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
TocopherolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Tocopherol
  »acetate
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
ViosterolT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NECT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »A
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »B NEC
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »B NEC
    »nicotinic acid
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »B1
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »B2
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »B6
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »B12
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »B15
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »C
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »D
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »D2
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »D3
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »E
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »E acetate
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »hematopoietic
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »K NEC
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »K1
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »K2
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »PP
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Vitamin NEC
  »ulceroprotectant
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
YeastT45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6
Yeast
  »dried
T45.2X1T45.2X2T45.2X3T45.2X4T45.2X5T45.2X6

Patient Education


Poisoning

A poison is any substance that is harmful to your body. You might swallow it, inhale it, inject it, or absorb it through your skin. Any substance can be poisonous if too much is taken. Poisons can include:

  • Prescription or over-the-counter medicines taken in doses that are too high
  • Overdoses of illegal drugs
  • Carbon monoxide from gas appliances
  • Household products, such as laundry powder or furniture polish
  • Pesticides
  • Indoor or outdoor plants
  • Metals such as lead and mercury

The effects of poisoning range from short-term illness to brain damage, coma, and death. To prevent poisoning it is important to use and store products exactly as their labels say. Keep dangerous products where children can't get to them. Treatment for poisoning depends on the type of poison. If you suspect someone has been poisoned, call your local poison control center at 1-800-222-1222 right away.


[Learn More in MedlinePlus]

Code History

  • FY 2024 - No Change, effective from 10/1/2023 through 9/30/2024
  • FY 2023 - No Change, effective from 10/1/2022 through 9/30/2023
  • FY 2022 - No Change, effective from 10/1/2021 through 9/30/2022
  • FY 2021 - No Change, effective from 10/1/2020 through 9/30/2021
  • FY 2020 - No Change, effective from 10/1/2019 through 9/30/2020
  • FY 2019 - No Change, effective from 10/1/2018 through 9/30/2019
  • FY 2018 - No Change, effective from 10/1/2017 through 9/30/2018
  • FY 2017 - No Change, effective from 10/1/2016 through 9/30/2017
  • FY 2016 - New Code, effective from 10/1/2015 through 9/30/2016. This was the first year ICD-10-CM was implemented into the HIPAA code set.

Footnotes

[1] Not chronic - A diagnosis code that does not fit the criteria for chronic condition (duration, ongoing medical treatment, and limitations) is considered not chronic. Some codes designated as not chronic are acute conditions. Other diagnosis codes that indicate a possible chronic condition, but for which the duration of the illness is not specified in the code description (i.e., we do not know the condition has lasted 12 months or longer) also are considered not chronic.