2024 ICD-10-CM Diagnosis Code G11.4

Hereditary spastic paraplegia

ICD-10-CM Code:
G11.4
ICD-10 Code for:
Hereditary spastic paraplegia
Is Billable?
Yes - Valid for Submission
Chronic Condition Indicator: [1]
Chronic
Code Navigator:

Code Classification

  • Diseases of the nervous system
    (G00–G99)
    • Systemic atrophies primarily affecting the central nervous system
      (G10-G14)
      • Hereditary ataxia
        (G11)

G11.4 is a billable diagnosis code used to specify a medical diagnosis of hereditary spastic paraplegia. The code is valid during the current fiscal year for the submission of HIPAA-covered transactions from October 01, 2023 through September 30, 2024.

Approximate Synonyms

The following clinical terms are approximate synonyms or lay terms that might be used to identify the correct diagnosis code:

  • Autosomal dominant complex hereditary spastic paraplegia
  • Autosomal dominant complex hereditary spastic paraplegia
  • Autosomal dominant complex hereditary spastic paraplegia
  • Autosomal dominant complex hereditary spastic paraplegia
  • Autosomal dominant complex hereditary spastic paraplegia
  • Autosomal dominant distal hereditary motor neuropathy
  • Autosomal dominant hereditary spastic paraplegia
  • Autosomal dominant spastic ataxia type 1
  • Autosomal dominant spastic paraplegia type 10
  • Autosomal dominant spastic paraplegia type 12
  • Autosomal dominant spastic paraplegia type 13
  • Autosomal dominant spastic paraplegia type 17
  • Autosomal dominant spastic paraplegia type 19
  • Autosomal dominant spastic paraplegia type 29
  • Autosomal dominant spastic paraplegia type 3
  • Autosomal dominant spastic paraplegia type 31
  • Autosomal dominant spastic paraplegia type 36
  • Autosomal dominant spastic paraplegia type 37
  • Autosomal dominant spastic paraplegia type 38
  • Autosomal dominant spastic paraplegia type 4
  • Autosomal dominant spastic paraplegia type 41
  • Autosomal dominant spastic paraplegia type 42
  • Autosomal dominant spastic paraplegia type 6
  • Autosomal dominant spastic paraplegia type 73
  • Autosomal dominant spastic paraplegia type 8
  • Autosomal dominant spastic paraplegia type 9A
  • Autosomal dominant spastic paraplegia type 9B
  • Autosomal recessive cerebellar ataxia with late-onset spasticity
  • Autosomal recessive complex spastic paraplegia due to Kennedy pathway dysfunction
  • Autosomal recessive hereditary spastic paraplegia
  • Autosomal recessive spastic ataxia of Charlevoix-Saguenay
  • Autosomal recessive spastic ataxia with leukoencephalopathy
  • Autosomal recessive spastic ataxia, optic atrophy, dysarthria syndrome
  • Autosomal recessive spastic paraplegia type 11
  • Autosomal recessive spastic paraplegia type 14
  • Autosomal recessive spastic paraplegia type 15
  • Autosomal recessive spastic paraplegia type 18
  • Autosomal recessive spastic paraplegia type 21
  • Autosomal recessive spastic paraplegia type 23
  • Autosomal recessive spastic paraplegia type 24
  • Autosomal recessive spastic paraplegia type 25
  • Autosomal recessive spastic paraplegia type 26
  • Autosomal recessive spastic paraplegia type 27
  • Autosomal recessive spastic paraplegia type 28
  • Autosomal recessive spastic paraplegia type 32
  • Autosomal recessive spastic paraplegia type 35
  • Autosomal recessive spastic paraplegia type 39
  • Autosomal recessive spastic paraplegia type 43
  • Autosomal recessive spastic paraplegia type 44
  • Autosomal recessive spastic paraplegia type 45
  • Autosomal recessive spastic paraplegia type 46
  • Autosomal recessive spastic paraplegia type 48
  • Autosomal recessive spastic paraplegia type 53
  • Autosomal recessive spastic paraplegia type 54
  • Autosomal recessive spastic paraplegia type 55
  • Autosomal recessive spastic paraplegia type 56
  • Autosomal recessive spastic paraplegia type 57
  • Autosomal recessive spastic paraplegia type 58
  • Autosomal recessive spastic paraplegia type 59
  • Autosomal recessive spastic paraplegia type 5A
  • Autosomal recessive spastic paraplegia type 60
  • Autosomal recessive spastic paraplegia type 61
  • Autosomal recessive spastic paraplegia type 62
  • Autosomal recessive spastic paraplegia type 63
  • Autosomal recessive spastic paraplegia type 64
  • Autosomal recessive spastic paraplegia type 66
  • Autosomal recessive spastic paraplegia type 67
  • Autosomal recessive spastic paraplegia type 69
  • Autosomal recessive spastic paraplegia type 70
  • Autosomal recessive spastic paraplegia type 71
  • Autosomal recessive spastic paraplegia type 74
  • Autosomal recessive spastic paraplegia type 75
  • Autosomal recessive spastic paraplegia type 76
  • Autosomal recessive spastic paraplegia type 77
  • Autosomal recessive spastic paraplegia type 78
  • Autosomal recessive spastic paraplegia type 9B
  • Autosomal spastic paraplegia type 30
  • Autosomal spastic paraplegia type 72
  • Bedouin spastic ataxia syndrome
  • Bilateral lower limb ataxia
  • Choreoathetosis
  • Chronic deafness
  • Complicated hereditary spastic paraplegia
  • Congenital miosis
  • Constricted pupil
  • Disorder of glutamine metabolism
  • Distal spinal muscular atrophy
  • Early-onset progressive encephalopathy, spastic ataxia, distal spinal muscular atrophy syndrome
  • Early-onset spastic ataxia, myoclonic epilepsy, neuropathy syndrome
  • Fryns macrocephaly
  • Hereditary sensory and autonomic neuropathy with spastic paraplegia
  • Hereditary spastic paraplegia
  • Infantile ascending hereditary spastic paralysis
  • Kyphoscoliosis, lateral tongue atrophy, hereditary spastic paraplegia syndrome
  • Macular corneal dystrophy
  • Maternally inherited mitochondrial deoxyribonucleic acid disease
  • Motor neuron disease due to hereditary spastic paraplegia
  • MT-ATP6-related mitochondrial spastic paraplegia
  • Palmoplantar keratoderma, spastic paralysis syndrome
  • Parkinsonism due to hereditary spastic paraplegia
  • Parkinsonism due to heredodegenerative disorder
  • Paroxysmal choreoathetosis
  • Paroxysmal dystonia
  • Paroxysmal dystonic choreoathetosis with episodic ataxia and spasticity
  • Progressive muscular atrophy
  • Punctate palmoplantar keratoderma
  • Pure hereditary spastic paraplegia
  • Second cranial nerve finding
  • Second cranial nerve finding
  • Second cranial nerve finding
  • Second cranial nerve finding
  • Second cranial nerve finding
  • Second cranial nerve finding
  • Second cranial nerve finding
  • Severe intellectual disability and progressive spastic paraplegia
  • Spastic ataxia with congenital miosis
  • Spastic ataxia, dysarthria due to glutaminase deficiency
  • Spastic paraparesis
  • Spastic paraparesis and deafness
  • Spastic paraplegia type 7
  • Spastic paraplegia with Paget disease of bone syndrome
  • Spastic paraplegia with precocious puberty syndrome
  • Spastic paraplegia, facial cutaneous lesion syndrome
  • Spastic paraplegia, glaucoma, intellectual disability syndrome
  • Spastic paraplegia, nephritis, deafness syndrome
  • Spastic paraplegia, neuropathy, poikiloderma syndrome
  • Spastic paraplegia, optic atrophy, neuropathy syndrome
  • Spastic paraplegia, severe developmental delay, epilepsy syndrome
  • SPOAN and SPOAN-related disorder
  • SPOAN and SPOAN-related disorder
  • SPOAN and SPOAN-related disorder
  • SPOAN and SPOAN-related disorder
  • SPOAN and SPOAN-related disorder
  • X-linked complex hereditary spastic paraplegia
  • X-linked hereditary spastic paraplegia
  • X-linked pure hereditary spastic paraplegia
  • X-linked spastic paraplegia type 16
  • X-linked spastic paraplegia type 2
  • X-linked spastic paraplegia type 34

Clinical Classification

Clinical Information

  • Macular Corneal Dystrophy

    a stromal corneal dystrophy, with autosomal recessive inheritance, that is caused by lack of or abnormal keratan sulfate.
  • Progressive Muscular Atrophy

    a rare, milder form of amyotrophic lateral sclerosis. it is characterized by a slowly progressive clinical course. signs and symptoms include muscle weakness, atrophy, and fasciculation.

Index to Diseases and Injuries References

The following annotation back-references for this diagnosis code are found in the injuries and diseases index. The Index to Diseases and Injuries is an alphabetical listing of medical terms, with each term mapped to one or more ICD-10-CM code(s).

Convert G11.4 to ICD-9-CM

  • ICD-9-CM Code: 334.1 - Hered spastic paraplegia
    Approximate Flag - The approximate mapping means there is not an exact match between the ICD-10 and ICD-9 codes and the mapped code is not a precise representation of the original code.

Patient Education


Neuromuscular Disorders

Neuromuscular disorders affect your neuromuscular system. They can cause problems with:

  • The nerves that control your muscles
  • Your muscles
  • Communication between your nerves and muscles

These disorders can cause your muscles to become weak and waste away. You may also have symptoms such as spasms, twitching, and pain.

Examples of neuromuscular disorders include:

  • Amyotrophic lateral sclerosis
  • Muscular dystrophy
  • Myasthenia gravis
  • Spinal muscular atrophy

There can be different causes for these diseases. Many of them are genetic.This means they are inherited (run in families) or are caused by a new mutation in your genes. Some neuromuscular disorders are autoimmune diseases. Sometimes the cause is unknown.

Many neuromuscular diseases have no cure. But treatments may improve symptoms, increase mobility, and lengthen life.


[Learn More in MedlinePlus]

Silver syndrome

Silver syndrome belongs to a group of genetic disorders known as hereditary spastic paraplegias. These disorders are characterized by progressive muscle stiffness (spasticity) and, frequently, development of paralysis of the lower limbs (paraplegia). Hereditary spastic paraplegias are divided into two types: pure and complex. Both types involve the lower limbs; the complex types may also involve the upper limbs, although to a lesser degree. In addition, the complex types may affect the brain and parts of the nervous system involved in muscle movement and sensations. Silver syndrome is a complex hereditary spastic paraplegia.

The first sign of Silver syndrome is usually weakness in the muscles of the hands. These muscles waste away (amyotrophy), resulting in abnormal positioning of the thumbs and difficulty using the fingers and hands for tasks such as handwriting. People with Silver syndrome often have high-arched feet (pes cavus) and spasticity in the legs. The signs and symptoms of Silver syndrome typically begin in late childhood but can start anytime from early childhood to late adulthood. The muscle problems associated with Silver syndrome slowly worsen with age, but affected individuals can remain active throughout life.


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Spastic paraplegia type 11

Spastic paraplegia type 11 is part of a group of genetic disorders known as hereditary spastic paraplegias. These disorders are characterized by progressive muscle stiffness (spasticity) and the development of paralysis of the lower limbs (paraplegia). Hereditary spastic paraplegias are divided into two types: pure and complex. The pure types involve the lower limbs. The complex types involve the lower limbs and can affect the upper limbs to a lesser degree. Complex spastic paraplegias also affect the structure or functioning of the brain and the peripheral nervous system, which consists of nerves connecting the brain and spinal cord to muscles and sensory cells that detect sensations such as touch, pain, heat, and sound. Spastic paraplegia type 11 is a complex hereditary spastic paraplegia.

Like all hereditary spastic paraplegias, spastic paraplegia type 11 involves spasticity of the leg muscles and muscle weakness. In almost all individuals with this type of spastic paraplegia, the tissue connecting the left and right halves of the brain (corpus callosum) is abnormally thin. People with this form of spastic paraplegia can also experience numbness, tingling, or pain in the arms and legs (sensory neuropathy); disturbance in the nerves used for muscle movement (motor neuropathy); intellectual disability; exaggerated reflexes (hyperreflexia) of the lower limbs; speech difficulties (dysarthria); reduced bladder control; and muscle wasting (amyotrophy). Less common features include difficulty swallowing (dysphagia), high-arched feet (pes cavus), an abnormal curvature of the spine (scoliosis), and involuntary movements of the eyes (nystagmus). The onset of symptoms varies greatly; however, abnormalities in muscle tone and difficulty walking usually become noticeable in adolescence.

Many features of spastic paraplegia type 11 are progressive. Most people experience a decline in intellectual ability and an increase in muscle weakness and nerve abnormalities over time. As the condition progresses, some people require wheelchair assistance.


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Spastic paraplegia type 15

Spastic paraplegia type 15 is part of a group of genetic disorders known as hereditary spastic paraplegias. These disorders are characterized by progressive muscle stiffness (spasticity) and the development of paralysis of the lower limbs (paraplegia). Spastic paraplegia type 15 is classified as a complex hereditary spastic paraplegia because it involves all four limbs as well as additional features, including abnormalities of the brain. In addition to the muscles and brain, spastic paraplegia type 15 affects the peripheral nervous system, which consists of nerves connecting the brain and spinal cord to muscles and sensory cells that detect sensations such as touch, pain, heat, and sound.

Spastic paraplegia type 15 usually becomes apparent in childhood or adolescence with the development of weak muscle tone (hypotonia), difficulty walking, or intellectual disability. In almost all affected individuals, the tissue connecting the left and right halves of the brain (corpus callosum) is abnormally thin and becomes thinner over time. Additionally, there is often a loss (atrophy) of nerve cells in several parts of the brain, including the cerebral cortex, which controls thinking and emotions, and the cerebellum, which coordinates movement.

People with this form of spastic paraplegia can have numbness, tingling, or pain in the arms and legs (sensory neuropathy); impairment of the nerves used for muscle movement (motor neuropathy); exaggerated reflexes (hyperreflexia) of the lower limbs; muscle wasting (amyotrophy); or reduced bladder control. Rarely, spastic paraplegia type 15 is associated with a group of movement abnormalities called parkinsonism, which includes tremors, rigidity, and unusually slow movement (bradykinesia). People with spastic paraplegia type 15 may have an eye condition called pigmentary maculopathy that often impairs vision. This condition results from the breakdown (degeneration) of tissue at the back of the eye called the macula, which is responsible for sharp central vision.

Most people with spastic paraplegia type 15 experience a decline in intellectual ability and an increase in muscle weakness and nerve abnormalities over time. As the condition progresses, many people require walking aids or wheelchair assistance in adulthood.


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Spastic paraplegia type 2

Spastic paraplegia type 2 is part of a group of genetic disorders known as hereditary spastic paraplegias. These disorders are characterized by progressive muscle stiffness (spasticity) and the development of paralysis of the lower limbs (paraplegia). Hereditary spastic paraplegias are divided into two types: pure and complex. The pure types involve the lower limbs. The complex types involve the lower limbs and can also affect the upper limbs to a lesser degree; the structure or functioning of the brain; and the nerves connecting the brain and spinal cord to muscles and sensory cells that detect sensations such as touch, pain, heat, and sound (the peripheral nervous system). Spastic paraplegia type 2 can occur in either the pure or complex form.

People with the pure form of spastic paraplegia type 2 experience spasticity in the lower limbs, usually without any additional features. People with the complex form of spastic paraplegia type 2 have lower limb spasticity and can also experience problems with movement and balance (ataxia); involuntary movements of the eyes (nystagmus); mild intellectual disability; involuntary, rhythmic shaking (tremor); and degeneration (atrophy) of the optic nerves, which carry information from the eyes to the brain. Symptoms usually become apparent between the ages of 1 and 5 years; those affected are typically able to walk and have a normal lifespan.


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Spastic paraplegia type 31

Spastic paraplegia type 31 is one of a group of genetic disorders known as hereditary spastic paraplegias. These disorders are characterized by progressive muscle stiffness (spasticity) and the development of paralysis of the lower limbs (paraplegia) caused by degeneration of nerve cells that trigger muscle movement (motor neurons). Hereditary spastic paraplegias are divided into two types: pure and complicated. The pure types involve only the lower limbs, while the complicated types also involve the upper limbs and other areas of the body, including the brain. Spastic paraplegia type 31 is usually a pure hereditary spastic paraplegia, although a few complicated cases have been reported.

The first signs and symptoms of spastic paraplegia type 31 usually appear before age 20 or after age 30. An early feature is difficulty walking due to spasticity and weakness, which typically affect both legs equally. People with spastic paraplegia type 31 can also experience progressive muscle wasting (amyotrophy) in the lower limbs, exaggerated reflexes (hyperreflexia), a decreased ability to feel vibrations, reduced bladder control, and high-arched feet (pes cavus). As the condition progresses, some individuals require walking support.


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Spastic paraplegia type 3A

Spastic paraplegia type 3A is one of a group of genetic disorders known as hereditary spastic paraplegias. These disorders are characterized by muscle stiffness (spasticity) and weakness in the lower limbs (paraplegia). Hereditary spastic paraplegias are often divided into two types: pure and complex. The pure types involve only the lower limbs, while the complex types also involve other areas of the body; additional features can include changes in vision, changes in intellectual functioning, difficulty walking, and disturbances in nerve function (neuropathy). Spastic paraplegia type 3A is usually a pure hereditary spastic paraplegia, although a few complex cases have been reported.

In addition to spasticity and weakness, which typically affect both legs equally, people with spastic paraplegia type 3A can also experience progressive muscle wasting (amyotrophy) in the lower limbs, reduced bladder control, an abnormal curvature of the spine (scoliosis), loss of sensation in the feet (peripheral neuropathy), or high arches of the feet (pes cavus). The signs and symptoms of spastic paraplegia type 3A usually appear before the age of 10; the average age of onset is 4 years. In some affected individuals the condition slowly worsens over time, sometimes leading to a need for walking support.


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Spastic paraplegia type 4

Spastic paraplegia type 4 (also known as SPG4) is the most common of a group of genetic disorders known as hereditary spastic paraplegias. These disorders are characterized by progressive muscle stiffness (spasticity) in the legs and difficulty walking. Hereditary spastic paraplegias are divided into two types: pure and complex. The pure types generally involve only spasticity of the lower limbs and walking difficulties. The complex types involve more widespread problems with the nervous system; the structure or functioning of the brain; and the nerves connecting the brain and spinal cord to muscles and sensory cells that detect sensations such as touch, pain, heat, and sound (the peripheral nervous system). In complex forms, there can also be features outside of the nervous system. Spastic paraplegia type 4 is usually a pure hereditary spastic paraplegia, although a few complex cases have been reported.

Like all hereditary spastic paraplegias, spastic paraplegia type 4 involves spasticity of the leg muscles and muscle weakness. People with this condition can also experience exaggerated reflexes (hyperreflexia), ankle spasms, high-arched feet (pes cavus), and reduced bladder control. Spastic paraplegia type 4 generally affects nerve and muscle function in the lower half of the body only.


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Spastic paraplegia type 5A

Spastic paraplegia type 5A is one of a group of genetic disorders known as hereditary spastic paraplegias. These disorders are characterized by muscle stiffness (spasticity) and severe weakness in the lower limbs (paraplegia). Hereditary spastic paraplegias are often divided into two types: pure and complex. The pure types involve spasticity and weakness only in the lower limbs, while the complex types involve additional problems with other areas of the body; additional features can include changes in vision, changes in intellectual functioning, brain abnormalities, and disturbances in nerve function (neuropathy). Spastic paraplegia type 5A is usually a pure hereditary spastic paraplegia, although complex type features have been reported in some individuals, usually in those who have had the condition for many years.

In addition to spasticity and weakness, people with spastic paraplegia type 5A can lose the ability to sense the position of their limbs or detect vibrations with their lower limbs. They may also have muscle wasting (amyotrophy), reduced bladder control, or high arches of the feet (pes cavus). The signs and symptoms of spastic paraplegia type 5A usually appear in adolescence but can begin at any time between infancy and mid-adulthood. The condition slowly worsens over time, often leading affected individuals to require walking support or wheelchair assistance.


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Spastic paraplegia type 7

Spastic paraplegia type 7 (also called SPG7) is one of more than 80 genetic disorders known as hereditary spastic paraplegias. These disorders primarily affect the brain and spinal cord (central nervous system), specifically nerve cells (neurons) that extend down the spinal cord. These neurons are used for muscle movement and sensation. Signs and symptoms of hereditary spastic paraplegias are characterized by progressive muscle stiffness (spasticity) in the legs and difficulty walking. 

Hereditary spastic paraplegias are divided into two types: pure and complex. The pure types generally involve only spasticity of the lower limbs and walking difficulties. The complex types involve more widespread problems with the nervous system; the structure or functioning of the brain; and the nerves connecting the brain and spinal cord to muscles and sensory cells that detect sensations such as touch, pain, heat, and sound (the peripheral nervous system). In complex forms, there can also be features outside of the nervous system. Spastic paraplegia type 7 can occur in either the pure or complex form.

Like all hereditary spastic paraplegias, spastic paraplegia type 7 involves spasticity of the leg muscles and some muscle weakness. People with this form of spastic paraplegia can also have ataxia; a pattern of movement abnormalities known as parkinsonism; exaggerated reflexes (hyperreflexia) in the arms; speech difficulties (dysarthria); difficulty swallowing (dysphagia); involuntary movements of the eyes (nystagmus); mild hearing loss; abnormal curvature of the spine (scoliosis); high-arched feet (pes cavus); numbness, tingling, or pain in the arms and legs (sensory neuropathy); disturbance in the nerves used for muscle movement (motor neuropathy); and muscle wasting (amyotrophy). The onset of symptoms varies greatly among those with spastic paraplegia type 7; however, abnormalities in muscle tone and other features usually become noticeable in adulthood.


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Spastic paraplegia type 8

Spastic paraplegia type 8 is part of a group of genetic disorders known as hereditary spastic paraplegias. These disorders are characterized by progressive muscle stiffness (spasticity) and the development of paralysis of the lower limbs (paraplegia). Hereditary spastic paraplegias are divided into two types: pure and complex. The pure types involve only the nerves and muscles controlling the lower limbs and bladder, whereas the complex types also have significant involvement of the nervous system in other parts of the body. Spastic paraplegia type 8 is a pure hereditary spastic paraplegia.

Like all hereditary spastic paraplegias, spastic paraplegia type 8 involves spasticity of the leg muscles and muscle weakness. People with this condition can also experience exaggerated reflexes (hyperreflexia), a decreased ability to feel vibrations, muscle wasting (amyotrophy), and reduced bladder control. The signs and symptoms of spastic paraplegia type 8 usually appear in early to mid-adulthood. As the muscle weakness and spasticity get worse, some people may need the aid of a cane, walker, or wheelchair.


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Troyer syndrome

Troyer syndrome is part of a group of genetic disorders known as hereditary spastic paraplegias. These disorders are characterized by progressive muscle stiffness (spasticity) and the development of paralysis of the lower limbs (paraplegia). Hereditary spastic paraplegias are divided into two types: pure and complex. The pure types involve the lower limbs. The complex types involve the lower limbs and can also affect the upper limbs to a lesser degree; the structure or functioning of the brain; and the nerves connecting the brain and spinal cord to muscles and sensory cells that detect sensations such as touch, pain, heat, and sound (the peripheral nervous system). Troyer syndrome is a complex hereditary spastic paraplegia.

People with Troyer syndrome can experience a variety of signs and symptoms. The most common characteristics of Troyer syndrome are spasticity of the leg muscles, progressive muscle weakness, paraplegia, muscle wasting in the hands and feet (distal amyotrophy), small stature, developmental delay, learning disorders, speech difficulties (dysarthria), and mood swings. Other characteristics can include exaggerated reflexes (hyperreflexia) in the lower limbs, uncontrollable movements of the limbs (choreoathetosis), skeletal abnormalities, and a bending outward (valgus) of the knees.

Troyer syndrome causes the degeneration and death of muscle cells and motor neurons (specialized nerve cells that control muscle movement) throughout a person's lifetime, leading to a slow progressive decline in muscle and nerve function. The severity of impairment related to Troyer syndrome increases as a person ages. Most affected individuals require a wheelchair by the time they are in their fifties or sixties.


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Code History

  • FY 2024 - No Change, effective from 10/1/2023 through 9/30/2024
  • FY 2023 - No Change, effective from 10/1/2022 through 9/30/2023
  • FY 2022 - No Change, effective from 10/1/2021 through 9/30/2022
  • FY 2021 - No Change, effective from 10/1/2020 through 9/30/2021
  • FY 2020 - No Change, effective from 10/1/2019 through 9/30/2020
  • FY 2019 - No Change, effective from 10/1/2018 through 9/30/2019
  • FY 2018 - No Change, effective from 10/1/2017 through 9/30/2018
  • FY 2017 - No Change, effective from 10/1/2016 through 9/30/2017
  • FY 2016 - New Code, effective from 10/1/2015 through 9/30/2016. This was the first year ICD-10-CM was implemented into the HIPAA code set.

Footnotes

[1] Chronic - a chronic condition code indicates a condition lasting 12 months or longer and its effect on the patient based on one or both of the following criteria:

  • The condition results in the need for ongoing intervention with medical products,treatment, services, and special equipment
  • The condition places limitations on self-care, independent living, and social interactions.