2024 ICD-10-CM Diagnosis Code T42.74XS

Poisoning by unspecified antiepileptic and sedative-hypnotic drugs, undetermined, sequela

ICD-10-CM Code:
T42.74XS
ICD-10 Code for:
Poisn by unsp antieplptc and sed-hypntc drugs, undet, sqla
Is Billable?
Yes - Valid for Submission
Chronic Condition Indicator: [1]
Not chronic
Code Navigator:

Code Classification

  • Injury, poisoning and certain other consequences of external causes
    (S00–T88)
    • Poisoning by, adverse effect of and underdosing of drugs, medicaments and biological substances
      (T36-T50)
      • Poisoning by, adverse effect of and underdosing of antiepileptic, sedative- hypnotic and antiparkinsonism drugs
        (T42)

T42.74XS is a billable diagnosis code used to specify a medical diagnosis of poisoning by unspecified antiepileptic and sedative-hypnotic drugs, undetermined, sequela. The code is valid during the current fiscal year for the submission of HIPAA-covered transactions from October 01, 2023 through September 30, 2024. The code is exempt from present on admission (POA) reporting for inpatient admissions to general acute care hospitals.

T42.74XS is a sequela code, includes a 7th character and should be used for complications that arise as a direct result of a condition like poisoning by unspecified antiepileptic and sedative-hypnotic drugs undetermined. According to ICD-10-CM Guidelines a "sequela" code should be used for chronic or residual conditions that are complications of an initial acute disease, illness or injury. The most common sequela is pain. Usually, two diagnosis codes are needed when reporting sequela. The first code describes the nature of the sequela while the second code describes the sequela or late effect.

Unspecified diagnosis codes like T42.74XS are acceptable when clinical information is unknown or not available about a particular condition. Although a more specific code is preferable, unspecified codes should be used when such codes most accurately reflect what is known about a patient's condition. Specific diagnosis codes should not be used if not supported by the patient's medical record.

Clinical Classification

Clinical CategoryCCSR Category CodeInpatient Default CCSROutpatient Default CCSR
Mental and substance use disorders; sequelaMBD034Y - Yes, default inpatient assignment for principal diagnosis or first-listed diagnosis.Y - Yes, default outpatient assignment for principal diagnosis or first-listed diagnosis.
Poisoning/toxic effect/adverse effects/underdosing, sequelaINJ075N - Not default inpatient assignment for principal diagnosis or first-listed diagnosis.N - Not default outpatient assignment for principal diagnosis or first-listed diagnosis.

Clinical Information

  • AIDS Arteritis, Central Nervous System

    inflammation of arteries in the central nervous system that occurs in patients with acquired immunodeficiency syndrome or aids-related opportunistic infections.
  • Brain Diseases

    pathologic conditions affecting the brain, which is composed of the intracranial components of the central nervous system. this includes (but is not limited to) the cerebral cortex; intracranial white matter; basal ganglia; thalamus; hypothalamus; brain stem; and cerebellum.
  • Brain Diseases, Metabolic

    acquired or inborn metabolic diseases that produce brain dysfunction or damage. these include primary (i.e., disorders intrinsic to the brain) and secondary (i.e., extracranial) metabolic conditions that adversely affect cerebral function.
  • Brain Diseases, Metabolic, Inborn

    brain disorders resulting from inborn metabolic errors, primarily from enzymatic defects which lead to substrate accumulation, product reduction, or increase in toxic metabolites through alternate pathways. the majority of these conditions are familial, however spontaneous mutation may also occur in utero.
  • Central Nervous System

    the main information-processing organs of the nervous system, consisting of the brain, spinal cord, and meninges.
  • Central Nervous System Agents

    a class of drugs producing both physiological and psychological effects through a variety of mechanisms. they can be divided into "specific" agents, e.g., affecting an identifiable molecular mechanism unique to target cells bearing receptors for that agent, and "nonspecific" agents, those producing effects on different target cells and acting by diverse molecular mechanisms. those with nonspecific mechanisms are generally further classed according to whether they produce behavioral depression or stimulation. those with specific mechanisms are classed by locus of action or specific therapeutic use. (from gilman ag, et al., goodman and gilman's the pharmacological basis of therapeutics, 8th ed, p252)
  • Central Nervous System Bacterial Infections

    bacterial infections of the brain, spinal cord, and meninges, including infections involving the perimeningeal spaces.
  • Central Nervous System Cysts

    congenital or acquired cysts of the brain, spinal cord, or meninges which may remain stable in size or undergo progressive enlargement.
  • Central Nervous System Depressants

    a very loosely defined group of drugs that tend to reduce the activity of the central nervous system. the major groups included here are ethyl alcohol, anesthetics, hypnotics and sedatives, narcotics, and tranquilizing agents (antipsychotics and antianxiety agents).
  • Central Nervous System Diseases

    diseases of any component of the brain (including the cerebral hemispheres, diencephalon, brain stem, and cerebellum) or the spinal cord.
  • Central Nervous System Fungal Infections

    mycoses of the brain, spinal cord, and meninges which may result in encephalitis; meningitis, fungal; myelitis; brain abscess; and epidural abscess. certain types of fungi may produce disease in immunologically normal hosts, while others are classified as opportunistic pathogens, causing illness primarily in immunocompromised individuals (e.g., acquired immunodeficiency syndrome).
  • Central Nervous System Helminthiasis

    infections of the brain; spinal cord; or meninges caused by helminths (parasitic worms).
  • Central Nervous System Infections

    pathogenic infections of the brain, spinal cord, and meninges. dna virus infections; rna virus infections; bacterial infections; mycoplasma infections; spirochaetales infections; fungal infections; protozoan infections; helminthiasis; and prion diseases may involve the central nervous system as a primary or secondary process.
  • Central Nervous System Neoplasms

    benign and malignant neoplastic processes that arise from or secondarily involve the brain, spinal cord, or meninges.
  • Central Nervous System Parasitic Infections

    infections of the brain, spinal cord, and meninges caused by parasites.
  • Central Nervous System Protozoal Infections

    infections of the brain, spinal cord, or meninges by single celled organisms of the former subkingdom known as protozoa. the central nervous system may be the primary or secondary site of protozoal infection. these diseases may occur as opportunistic infections or arise in immunocompetent hosts.
  • Central Nervous System Sensitization

    an increased response to stimulation that is mediated by amplification of signaling in the central nervous system (cns).
  • Central Nervous System Stimulants

    a loosely defined group of drugs that tend to increase behavioral alertness, agitation, or excitation. they work by a variety of mechanisms, but usually not by direct excitation of neurons. the many drugs that have such actions as side effects to their main therapeutic use are not included here.
  • Central Nervous System Vascular Malformations

    congenital, inherited, or acquired abnormalities involving arteries; veins; or venous sinuses in the brain; spinal cord; and meninges.
  • Central Nervous System Venous Angioma

    a vascular anomaly characterized by a radial or wedge-shaped arrangement of dilated veins draining into a larger vein in the brain, spinal cord, or the meninges. veins in a venous angioma are surrounded by normal nervous tissue, unlike a central nervous system cavernous hemangioma that lacks intervening nervous tissue. drainage of venous angioma is fully integrated with the body's venous system, therefore, in most cases there is no clinical signs and rare bleeding.
  • Central Nervous System Viral Diseases

    viral infections of the brain, spinal cord, meninges, or perimeningeal spaces.
  • Cerebral Phaeohyphomycosis

    cns infections caused by neurotropic dematiaceous fungi that contain melanin in their cell walls. the infections often result in brain abscess; encephalitis; and meningitis in patients who are often immunocompetent. the common causative fungi include members cladophialophora bantiana, exophiala dermatitidis, rhinocladiella mackenziei, and ochroconis gallopavum. r. mackenziei infection is seen almost exclusively in patients from the middle east.
  • Hemangioma, Cavernous, Central Nervous System

    a vascular anomaly composed of a collection of large, thin walled tortuous veins that can occur in any part of the central nervous system but lack intervening nervous tissue. familial occurrence is common and has been associated with a number of genes mapped to 7q, 7p and 3q. clinical features include seizures; headache; stroke; and progressive neurological deficit.
  • Hereditary Central Nervous System Demyelinating Diseases

    inherited conditions characterized by a loss of myelin in the central nervous system.
  • Lupus Vasculitis, Central Nervous System

    central nervous system vasculitis that is associated with systemic lupus erythematosus. clinical manifestations may include dementia; seizures; cranial nerve diseases; hemiparesis; blindness; dysphasia; and other neurological disorders.
  • Lyme Neuroborreliosis

    nervous system infections caused by tick-borne spirochetes of the borrelia burgdorferi group. the disease may affect elements of the central or peripheral nervous system in isolation or in combination. common clinical manifestations include a lymphocytic meningitis, cranial neuropathy (most often a facial neuropathy), polyradiculopathy, and a mild loss of memory and other cognitive functions. less often more extensive inflammation involving the central nervous system (encephalomyelitis) may occur. in the peripheral nervous system, b. burgdorferi infection is associated with mononeuritis multiplex and polyradiculoneuritis. (from j neurol sci 1998 jan 8;153(2):182-91)
  • Neurocysticercosis

    infection of the brain, spinal cord, or perimeningeal structures with the larval forms of the genus taenia (primarily t. solium in humans). lesions formed by the organism are referred to as cysticerci. the infection may be subacute or chronic, and the severity of symptoms depends on the severity of the host immune response and the location and number of lesions. seizures represent the most common clinical manifestation although focal neurologic deficits may occur. (from joynt, clinical neurology, 1998, ch27, pp46-50)
  • Neuroschistosomiasis

    schistosomiasis of the brain, spinal cord, or meninges caused by infections with trematodes of the genus schistosoma (primarily schistosoma japonicum; schistosoma mansoni; and schistosoma haematobium in humans). s. japonicum infections of the nervous system may cause an acute meningoencephalitis or a chronic encephalopathy. s. mansoni and s. haematobium nervous system infections are associated with acute transverse myelitis involving the lower portions of the spinal cord. (from joynt, clinical neurology, 1998, ch27, pp61-2)
  • Neurosyphilis

    infections of the central nervous system caused by treponema pallidum which present with a variety of clinical syndromes. the initial phase of infection usually causes a mild or asymptomatic meningeal reaction. the meningovascular form may present acutely as brain infarction. the infection may also remain subclinical for several years. late syndromes include general paresis; tabes dorsalis; meningeal syphilis; syphilitic optic atrophy; and spinal syphilis. general paresis is characterized by progressive dementia; dysarthria; tremor; myoclonus; seizures; and argyll-robertson pupils. (adams et al., principles of neurology, 6th ed, pp722-8)
  • Toxoplasmosis, Cerebral

    infections of the brain caused by the protozoan toxoplasma gondii that primarily arise in individuals with immunologic deficiency syndromes (see also aids-related opportunistic infections). the infection may involve the brain diffusely or form discrete abscesses. clinical manifestations include seizures, altered mentation, headache, focal neurologic deficits, and intracranial hypertension. (from joynt, clinical neurology, 1998, ch27, pp41-3)
  • Tuberculosis, Central Nervous System

    tuberculosis of the brain, spinal cord, or meninges (tuberculosis, meningeal), most often caused by mycobacterium tuberculosis and rarely by mycobacterium bovis. the infection may be limited to the nervous system or coexist in other organs (e.g., tuberculosis, pulmonary). the organism tends to seed the meninges causing a diffuse meningitis and leads to the formation of tuberculoma, which may occur within the brain, spinal cord, or perimeningeal spaces. tuberculous involvement of the vertebral column (tuberculosis, spinal) may result in nerve root or spinal cord compression. (from adams et al., principles of neurology, 6th ed, pp717-20)
  • Vasculitis, Central Nervous System

    inflammation of blood vessels within the central nervous system. primary vasculitis is usually caused by autoimmune or idiopathic factors, while secondary vasculitis is caused by existing disease process. clinical manifestations are highly variable but include headache; seizures; behavioral alterations; intracranial hemorrhages; transient ischemic attack; and brain infarction. (from adams et al., principles of neurology, 6th ed, pp856-61)
  • Vertigo

    an illusion of movement, either of the external world revolving around the individual or of the individual revolving in space. vertigo may be associated with disorders of the inner ear (ear, inner); vestibular nerve; brainstem; or cerebral cortex. lesions in the temporal lobe and parietal lobe may be associated with focal seizures that may feature vertigo as an ictal manifestation. (from adams et al., principles of neurology, 6th ed, pp300-1)

Coding Guidelines

When coding a poisoning or reaction to the improper use of a medication (e.g., overdose, wrong substance given or taken in error, wrong route of administration), first assign the appropriate code from categories T36-T50. The poisoning codes have an associated intent as their 5th or 6th character (accidental, intentional self-harm, assault and undetermined. If the intent of the poisoning is unknown or unspecified, code the intent as accidental intent. The undetermined intent is only for use if the documentation in the record specifies that the intent cannot be determined. Use additional code(s) for all manifestations of poisonings.

The appropriate 7th character is to be added to each code from block Poisoning by, adverse effect of and underdosing of antiepileptic, sedative- hypnotic and antiparkinsonism drugs (T42). Use the following options for the aplicable episode of care:

  • A - initial encounter
  • D - subsequent encounter
  • S - sequela

Present on Admission (POA)

T42.74XS is exempt from POA reporting - The Present on Admission (POA) indicator is used for diagnosis codes included in claims involving inpatient admissions to general acute care hospitals. POA indicators must be reported to CMS on each claim to facilitate the grouping of diagnoses codes into the proper Diagnostic Related Groups (DRG). CMS publishes a listing of specific diagnosis codes that are exempt from the POA reporting requirement. Review other POA exempt codes here.

CMS POA Indicator Options and Definitions

POA IndicatorReason for CodeCMS will pay the CC/MCC DRG?
YDiagnosis was present at time of inpatient admission.YES
NDiagnosis was not present at time of inpatient admission.NO
UDocumentation insufficient to determine if the condition was present at the time of inpatient admission.NO
WClinically undetermined - unable to clinically determine whether the condition was present at the time of inpatient admission.YES
1Unreported/Not used - Exempt from POA reporting. NO

Convert T42.74XS to ICD-9-CM

  • ICD-9-CM Code: 909.0 - Late eff drug poisoning
    Combination Flag - Multiple codes are needed to describe the source diagnosis code. Correct coding should be done based on contextual judgment.
  • ICD-9-CM Code: E989 - Late eff inj-undet circ
    Combination Flag - Multiple codes are needed to describe the source diagnosis code. Correct coding should be done based on contextual judgment.

Table of Drugs and Chemicals

The parent code T42.74 of the current diagnosis code is referenced in the Table of Drugs and Chemicals, this table contains a classification of drugs, industrial solvents, corrosive gases, noxious plants, pesticides, and other toxic agents.

According to ICD-10-CM coding guidelines it is advised to do not code directly from the Table of Drugs and Chemicals, instead always refer back to the Tabular List when doing the initial coding. Each substance in the table is assigned a code according to the poisoning classification and external causes of adverse effects. It is important to use as many codes as necessary to specify all reported drugs, medicinal or chemical substances. If the same diagnosis code describes the causative agent for more than one adverse reaction, poisoning, toxic effect or underdosing, utilize the code only once.

Substance Poisoning
Accidental
(unintentional)
Poisoning
Accidental
(self-harm)
Poisoning
Assault
Poisoning
Undetermined
Adverse
effect
Underdosing
AnticonvulsantT42.71T42.72T42.73T42.74T42.75T42.76
Anticonvulsant
  »barbiturate
T42.71T42.72T42.73T42.74T42.75T42.76
Anticonvulsant
  »combination (with barbiturate)
T42.71T42.72T42.73T42.74T42.75T42.76
Anticonvulsant
  »hydantoin
T42.71T42.72T42.73T42.74T42.75T42.76
Anticonvulsant
  »hypnotic NEC
T42.71T42.72T42.73T42.74T42.75T42.76
Anticonvulsant
  »oxazolidinedione
T42.71T42.72T42.73T42.74T42.75T42.76
Anticonvulsant
  »pyrimidinedione
T42.71T42.72T42.73T42.74T42.75T42.76
Anticonvulsant
  »specified NEC
T42.71T42.72T42.73T42.74T42.75T42.76
Anticonvulsant
  »succinimide
T42.71T42.72T42.73T42.74T42.75T42.76
Antiepilepsy agentT42.71T42.72T42.73T42.74T42.75T42.76
Antiepilepsy agent
  »combination
T42.71T42.72T42.73T42.74T42.75T42.76
Antiepilepsy agent
  »mixed
T42.71T42.72T42.73T42.74T42.75T42.76
Antiepilepsy agent
  »specified, NEC
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous systemT42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »anesthetic (general) NEC
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »anesthetic (general) NEC
      »gases NEC
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »anesthetic (general) NEC
      »intravenous
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »barbiturates
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »benzodiazepines
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »bromides
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »cannabis sativa
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »chloral hydrate
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »ethanol
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »hallucinogenics
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »hypnotics
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »hypnotics
      »specified NEC
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »muscle relaxants
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »paraldehyde
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »sedatives; sedative-hypnotics
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »sedatives; sedative-hypnotics
      »mixed NEC
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »depressants
    »sedatives; sedative-hypnotics
      »specified NEC
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »muscle-tone depressants
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »stimulants
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »stimulants
    »amphetamines
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »stimulants
    »analeptics
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »stimulants
    »antidepressants
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »stimulants
    »opiate antagonists
T42.71T42.72T42.73T42.74T42.75T42.76
Central nervous system
  »stimulants
    »specified NEC
T42.71T42.72T42.73T42.74T42.75T42.76
HypnoticT42.71T42.72T42.73T42.74T42.75T42.76
Hypnotic
  »anticonvulsant
T42.71T42.72T42.73T42.74T42.75T42.76
Hypnotic
  »specified NEC
T42.71T42.72T42.73T42.74T42.75T42.76
Sedative NECT42.71T42.72T42.73T42.74T42.75T42.76
Sedative NEC
  »mixed NEC
T42.71T42.72T42.73T42.74T42.75T42.76
Sleeping draught, pillT42.71T42.72T42.73T42.74T42.75T42.76
SoporificT42.71T42.72T42.73T42.74T42.75T42.76
Soporific drugT42.71T42.72T42.73T42.74T42.75T42.76
Soporific drug
  »specified type NEC
T42.71T42.72T42.73T42.74T42.75T42.76

Patient Education


Poisoning

A poison is any substance that is harmful to your body. You might swallow it, inhale it, inject it, or absorb it through your skin. Any substance can be poisonous if too much is taken. Poisons can include:

  • Prescription or over-the-counter medicines taken in doses that are too high
  • Overdoses of illegal drugs
  • Carbon monoxide from gas appliances
  • Household products, such as laundry powder or furniture polish
  • Pesticides
  • Indoor or outdoor plants
  • Metals such as lead and mercury

The effects of poisoning range from short-term illness to brain damage, coma, and death. To prevent poisoning it is important to use and store products exactly as their labels say. Keep dangerous products where children can't get to them. Treatment for poisoning depends on the type of poison. If you suspect someone has been poisoned, call your local poison control center at 1-800-222-1222 right away.


[Learn More in MedlinePlus]

Code History

  • FY 2024 - No Change, effective from 10/1/2023 through 9/30/2024
  • FY 2023 - No Change, effective from 10/1/2022 through 9/30/2023
  • FY 2022 - No Change, effective from 10/1/2021 through 9/30/2022
  • FY 2021 - No Change, effective from 10/1/2020 through 9/30/2021
  • FY 2020 - No Change, effective from 10/1/2019 through 9/30/2020
  • FY 2019 - No Change, effective from 10/1/2018 through 9/30/2019
  • FY 2018 - No Change, effective from 10/1/2017 through 9/30/2018
  • FY 2017 - No Change, effective from 10/1/2016 through 9/30/2017
  • FY 2016 - New Code, effective from 10/1/2015 through 9/30/2016. This was the first year ICD-10-CM was implemented into the HIPAA code set.

Footnotes

[1] Not chronic - A diagnosis code that does not fit the criteria for chronic condition (duration, ongoing medical treatment, and limitations) is considered not chronic. Some codes designated as not chronic are acute conditions. Other diagnosis codes that indicate a possible chronic condition, but for which the duration of the illness is not specified in the code description (i.e., we do not know the condition has lasted 12 months or longer) also are considered not chronic.